Molecular mechanisms of heterogeneous oligomerization of huntingtin proteins
Published 2019 View Full Article
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Title
Molecular mechanisms of heterogeneous oligomerization of huntingtin proteins
Authors
Keywords
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Journal
Scientific Reports
Volume 9, Issue 1, Pages -
Publisher
Springer Science and Business Media LLC
Online
2019-05-20
DOI
10.1038/s41598-019-44151-0
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- Prion-like transmission of neuronal huntingtin aggregates to phagocytic glia in the Drosophila brain
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- Mutant huntingtin is present in neuronal grafts in huntington disease patients
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- (2012) S. Piana et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Secondary Structures of Native and Pathogenic Huntingtin N-Terminal Fragments
- (2011) Maciej Długosz et al. JOURNAL OF PHYSICAL CHEMISTRY B
- Critical nucleus size for disease-related polyglutamine aggregation is repeat-length dependent
- (2011) Karunakar Kar et al. NATURE STRUCTURAL & MOLECULAR BIOLOGY
- Inhibiting the ubiquitin–proteasome system leads to preferential accumulation of toxic N-terminal mutant huntingtin fragments
- (2010) Xiang Li et al. HUMAN MOLECULAR GENETICS
- Polyglutamine Induced Misfolding of Huntingtin Exon1 is Modulated by the Flanking Sequences
- (2010) Vinal V. Lakhani et al. PLoS Computational Biology
- GROMACS 4: Algorithms for Highly Efficient, Load-Balanced, and Scalable Molecular Simulation
- (2008) Berk Hess et al. Journal of Chemical Theory and Computation
- Ab Initio Folding of Proteins with All-Atom Discrete Molecular Dynamics
- (2008) Feng Ding et al. STRUCTURE
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