O-GlcNAcylation of protein kinase A catalytic subunits enhances its activity: a mechanism linked to learning and memory deficits in Alzheimer's disease
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Title
O-GlcNAcylation of protein kinase A catalytic subunits enhances its activity: a mechanism linked to learning and memory deficits in Alzheimer's disease
Authors
Keywords
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Journal
AGING CELL
Volume 15, Issue 3, Pages 455-464
Publisher
Wiley
Online
2016-02-04
DOI
10.1111/acel.12449
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- (2013) N. Jin et al. NUCLEIC ACIDS RESEARCH
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- Dynamic O-GlcNAc modification regulates CREB-mediated gene expression and memory formation
- (2012) Jessica E Rexach et al. Nature Chemical Biology
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- (2012) Susan S. Taylor et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- O-linked β-N-acetylglucosaminidase inhibitor attenuates β-amyloid plaque and rescues memory impairment
- (2012) Chaeyoung Kim et al. NEUROBIOLOGY OF AGING
- The molecular biology of memory: cAMP, PKA, CRE, CREB-1, CREB-2, and CPEB
- (2012) Eric R Kandel Molecular Brain
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- (2011) Jianhua Shi et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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- (2009) F. Liu et al. BRAIN
- Degradation of cAMP-Responsive Element-Binding Protein by the Ubiquitin-Proteasome Pathway Contributes to Glucotoxicity in -Cells and Human Pancreatic Islets
- (2009) S. Costes et al. DIABETES
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- (2009) Jon M. Steichen et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Enhanced CREB-dependent gene expression increases the excitability of neurons in the basal amygdala and primes the consolidation of contextual and cued fear memory
- (2009) J. Viosca et al. LEARNING & MEMORY
- CREB regulates excitability and the allocation of memory to subsets of neurons in the amygdala
- (2009) Yu Zhou et al. NATURE NEUROSCIENCE
- In VivoModulation ofO-GlcNAc Levels Regulates Hippocampal Synaptic Plasticity through Interplay with Phosphorylation
- (2008) Melanie K. Tallent et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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- (2007) Minseok Song et al. CELLULAR SIGNALLING
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