Redox-Mediated Angiogenesis in the Hypoxic Joint of Inflammatory Arthritis

Hypoxia: how does the monocyte-macrophage system respond to changes in oxygen availability?

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Hypoxia induces nitric oxide synthase in rheumatoid synoviocytes: consequences on NADPH oxidase regulation

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Lipid Peroxidation Product 4-Hydroxy-trans-2-nonenal Causes Endothelial Activation by Inducing Endoplasmic Reticulum Stress

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The HIF1 target gene NOX2 promotes angiogenesis through urotensin-II

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Hypoxia—a key regulator of angiogenesis and inflammation in rheumatoid arthritis

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Redox balance dynamically regulates vascular growth and remodeling

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Tumor necrosis factor blocking therapy alters joint inflammation and hypoxia

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The expression and function of vascular endothelial growth factor in retinal pigment epithelial (RPE) cells is regulated by 4-hydroxynonenal (HNE) and glutathione S-transferaseA4-4

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Targeting Vascular NADPH Oxidase 1 Blocks Tumor Angiogenesis through a PPARα Mediated Mechanism

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Differential Expression of NK Receptors CD94 and NKG2A by T Cells in Rheumatoid Arthritis Patients in Remission Compared to Active Disease

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Blockade of Toll-like receptor 2 prevents spontaneous cytokine release from rheumatoid arthritis ex vivo synovial explant cultures

(2011) Sinéad Nic An Ultaigh et al.   ARTHRITIS RESEARCH & THERAPY

Successful tumour necrosis factor (TNF) blocking therapy suppresses oxidative stress and hypoxia-induced mitochondrial mutagenesis in inflammatory arthritis

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Synovial tissue hypoxia and inflammation in vivo

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Angiogenesis and blood vessel stability in inflammatory arthritis

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The transcriptional response of normal and rheumatoid arthritis synovial fibroblasts to hypoxia

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NADPH oxidase-derived reactive oxygen species: involvement in vascular physiology and pathology

(2010) Adrian Manea   CELL AND TISSUE RESEARCH

Angiogenesis and vasculogenesis in rheumatoid arthritis

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5-Lipoxygenase plays an essential role in 4-HNE-enhanced ROS production in murine macrophages via activation of NADPH oxidase

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Oxidative stress induces angiogenesis by activating TLR2 with novel endogenous ligands

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Oxidative damage in synovial tissue is associated with in vivo hypoxic status in the arthritic joint

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Hypoxia Activates NF-κB–Dependent Gene Expression Through the Canonical Signaling Pathway

(2009) Kathryn M. Oliver et al.   ANTIOXIDANTS & REDOX SIGNALING

Aberrant Reactive Oxygen and Nitrogen Species Generation in Rheumatoid Arthritis (RA): Causes and Consequences for Immune Function, Cell Survival, and Therapeutic Intervention

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Angiogenesis in rheumatoid arthritis

(2009) Zoltán Szekanecz et al.   AUTOIMMUNITY

The role of hypoxia and HIF-dependent signalling events in rheumatoid arthritis

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Hypoxia upregulates angiogenesis and synovial cell migration in rheumatoid arthritis

(2009) Mohammed A Akhavani et al.   ARTHRITIS RESEARCH & THERAPY

NADPH Oxidase-Dependent Signaling in Endothelial Cells: Role in Physiology and Pathophysiology

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Differential roles of PDGFR-α and PDGFR-β in angiogenesis and vessel stability

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Synovial Hypoxia as a Cause of Tendon Rupture in Rheumatoid Arthritis

(2008) Branavan Sivakumar et al.   JOURNAL OF HAND SURGERY-AMERICAN VOLUME