Honokiol protects against doxorubicin cardiotoxicity via improving mitochondrial function in mouse hearts
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Title
Honokiol protects against doxorubicin cardiotoxicity via improving mitochondrial function in mouse hearts
Authors
Keywords
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Journal
Scientific Reports
Volume 7, Issue 1, Pages -
Publisher
Springer Nature
Online
2017-09-14
DOI
10.1038/s41598-017-12095-y
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- Honokiol Decreases Lung Cancer Metastasis through Inhibition of the STAT3 Signaling Pathway
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- (2015) Todd J. Kilbaugh et al. EXPERIMENTAL NEUROLOGY
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- (2015) YONGWU YU et al. Molecular Medicine Reports
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- Honokiol: A non-adipogenic PPARγ agonist from nature
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- Small molecule kinase inhibitors block the ZAK-dependent inflammatory effects of doxorubicin
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- Doxorubicin-Induced Cardiotoxicity: From Bioenergetic Failure and Cell Death to Cardiomyopathy
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- Quercetin Inhibits Left Ventricular Hypertrophy in Spontaneously Hypertensive Rats and Inhibits Angiotensin II-Induced H9C2 Cells Hypertrophy by Enhancing PPAR-γ Expression and Suppressing AP-1 Activity
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- Carnitine Palmitoyltransferase-1b Deficiency Aggravates Pressure Overload–Induced Cardiac Hypertrophy Caused by Lipotoxicity
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- Doxorubicin-induced cardiomyopathy: From molecular mechanisms to therapeutic strategies
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- Comparison of antioxidant abilities of magnolol and honokiol to scavenge radicals and to protect DNA
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- Activation of peroxisome proliferator-activated receptor-γ downregulates soluble epoxide hydrolase in cardiomyocytes
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- Peroxisome Proliferator-Activated Receptor β/δ Activation in Adult Hearts Facilitates Mitochondrial Function and Cardiac Performance Under Pressure-Overload Condition
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- Effects of Chronic Treatment with Honokiol in Spontaneously Hypertensive Rats
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- Differences in doxorubicin-induced apoptotic signaling in adult and immature cardiomyocytes
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