Dysregulated Tim-3 expression on natural killer cells is associated with increased Galectin-9 levels in HIV-1 infection
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Title
Dysregulated Tim-3 expression on natural killer cells is associated with increased Galectin-9 levels in HIV-1 infection
Authors
Keywords
Tim-3, Gal-9, HIV-1, Innate immunity, NK cells
Journal
Retrovirology
Volume 10, Issue 1, Pages 74
Publisher
Springer Nature
Online
2013-07-19
DOI
10.1186/1742-4690-10-74
References
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Related references
Note: Only part of the references are listed.- HIV Infection Deregulates Tim-3 Expression on Innate Cells
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- Galectin-9 Functionally Impairs Natural Killer Cells in Humans and Mice
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- TIM-3 Does Not Act as a Receptor for Galectin-9
- (2013) Judith Leitner et al. PLoS Pathogens
- Tim-3 marks human natural killer cell maturation and suppresses cell-mediated cytotoxicity
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- Tim-3 is an inducible human natural killer cell receptor that enhances interferon gamma production in response to galectin-9
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- Tim-3/galectin-9 signaling pathway mediates T-cell dysfunction and predicts poor prognosis in patients with hepatitis B virus-associated hepatocellular carcinoma
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- Tim-3 Negatively Regulates Cytotoxicity in Exhausted CD8+ T Cells in HIV Infection
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- HIV-1 adaptation to NK-cell-mediated immune pressure
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- Cross-Sectional Detection of Acute HIV Infection: Timing of Transmission, Inflammation and Antiretroviral Therapy
- (2011) Cynthia Gay et al. PLoS One
- Galectin-9 binding to cell surface protein disulfide isomerase regulates the redox environment to enhance T-cell migration and HIV entry
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- HCV-specific T cells in HCV/HIV co-infection show elevated frequencies of dual Tim-3/PD-1 expression that correlate with liver disease progression
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- Innate immunity defines the capacity of antiviral T cells to limit persistent infection
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- T cell immunoglobulin- and mucin-domain-containing molecule-3 (Tim-3) mediates natural killer cell suppression in chronic hepatitis B
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- HIV Protective KIR3DL1 and HLA-B Genotypes Influence NK Cell Function Following Stimulation with HLA-Devoid Cells
- (2010) Salix Boulet et al. JOURNAL OF IMMUNOLOGY
- T Cell/Transmembrane, Ig, and Mucin-3 Allelic Variants Differentially Recognize Phosphatidylserine and Mediate Phagocytosis of Apoptotic Cells
- (2010) Rosemarie H. DeKruyff et al. JOURNAL OF IMMUNOLOGY
- A combined genotype of KIR3DL1 high expressing alleles and HLA-B*57 is associated with a reduced risk of HIV infection
- (2009) Salix Boulet et al. AIDS
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- (2009) Salix Boulet et al. AIDS
- IL-21 augments natural killer effector functions in chronically HIV-infected individuals
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- Tim-3 mediates phagocytosis of apoptotic cells and cross-presentation
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- HLA Class I Subtype-Dependent Expansion of KIR3DS1+ and KIR3DL1+ NK Cells during Acute Human Immunodeficiency Virus Type 1 Infection
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- Role of Natural Killer Cells in a Cohort of Elite Suppressors: Low Frequency of the Protective KIR3DS1 Allele and Limited Inhibition of Human Immunodeficiency Virus Type 1 Replication In Vitro
- (2009) K. A. O'Connell et al. JOURNAL OF VIROLOGY
- Induction of a Striking Systemic Cytokine Cascade prior to Peak Viremia in Acute Human Immunodeficiency Virus Type 1 Infection, in Contrast to More Modest and Delayed Responses in Acute Hepatitis B and C Virus Infections
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- Negative Immune Regulator Tim-3 Is Overexpressed on T Cells in Hepatitis C Virus Infection and Its Blockade Rescues Dysfunctional CD4+ and CD8+ T Cells
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- Structural Features of Galectin-9 and Galectin-1 That Determine Distinct T Cell Death Pathways
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- Tim-3 expression defines a novel population of dysfunctional T cells with highly elevated frequencies in progressive HIV-1 infection
- (2008) R. Brad Jones et al. JOURNAL OF EXPERIMENTAL MEDICINE
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