PYK2 sustains endosomal-derived receptor signalling and enhances epithelial-to-mesenchymal transition
Published 2015 View Full Article
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Title
PYK2 sustains endosomal-derived receptor signalling and enhances epithelial-to-mesenchymal transition
Authors
Keywords
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Journal
Nature Communications
Volume 6, Issue 1, Pages -
Publisher
Springer Nature
Online
2015-02-04
DOI
10.1038/ncomms7064
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Note: Only part of the references are listed.- Surgery-induced wound response promotes stem-like and tumor-initiating features of breast cancer cells, via STAT3 signaling
- (2015) Ilenia Segatto et al. Oncotarget
- The lipid-transfer protein Nir2 enhances epithelial-mesenchymal transition and facilitates breast cancer metastasis
- (2014) O. Keinan et al. JOURNAL OF CELL SCIENCE
- Molecular mechanisms of epithelial–mesenchymal transition
- (2014) Samy Lamouille et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- High MET expression is an adverse prognostic factor in patients with triple-negative breast cancer
- (2013) F Zagouri et al. BRITISH JOURNAL OF CANCER
- Increased expression of Slug and Vimentin as novel predictive biomarkers for lymph node metastasis and poor prognosis in colorectal cancer
- (2013) Yuji Toiyama et al. CARCINOGENESIS
- The phosphatidylinositol-transfer protein Nir2 binds phosphatidic acid and positively regulates phosphoinositide signalling
- (2013) SoHui Kim et al. EMBO REPORTS
- MET is a potential target for use in combination therapy with EGFR inhibition in triple-negative/basal-like breast cancer
- (2013) Yu Jin Kim et al. INTERNATIONAL JOURNAL OF CANCER
- Hic-5 promotes invadopodia formation and invasion during TGF-β–induced epithelial–mesenchymal transition
- (2012) Jeanine Pignatelli et al. JOURNAL OF CELL BIOLOGY
- TGF-β stimulates Pyk2 expression as part of an epithelial-mesenchymal transition program required for metastatic outgrowth of breast cancer
- (2012) M K Wendt et al. ONCOGENE
- Non-Stimulated, Agonist-Stimulated and Store-Operated Ca2+ Influx in MDA-MB-468 Breast Cancer Cells and the Effect of EGF-Induced EMT on Calcium Entry
- (2012) Felicity M. Davis et al. PLoS One
- Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies
- (2011) Brian D. Lehmann et al. JOURNAL OF CLINICAL INVESTIGATION
- Temporal and Spatial Cooperation of Snail1 and Twist1 during Epithelial-Mesenchymal Transition Predicts for Human Breast Cancer Recurrence
- (2011) D. D. Tran et al. MOLECULAR CANCER RESEARCH
- Quantitative analysis of transient and sustained transforming growth factor- signaling dynamics
- (2011) Z. Zi et al. Molecular Systems Biology
- HGF-independent potentiation of EGFR action by c-Met
- (2011) A M Dulak et al. ONCOGENE
- Proline-Rich Tyrosine Kinase 2 (Pyk2) Promotes Cell Motility of Hepatocellular Carcinoma through Induction of Epithelial to Mesenchymal Transition
- (2011) Chris K. Sun et al. PLoS One
- An overview of the c-MET signaling pathway
- (2011) Shawna Leslie Organ et al. Therapeutic Advances in Medical Oncology
- Structural Requirements for VAP-B Oligomerization and Their Implication in Amyotrophic Lateral Sclerosis-associated VAP-B(P56S) Neurotoxicity
- (2010) SoHui Kim et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Cellular functions of FAK kinases: insight into molecular mechanisms and novel functions
- (2010) M. D. Schaller JOURNAL OF CELL SCIENCE
- ErbB/EGF Signaling and EMT in Mammary Development and Breast Cancer
- (2010) Katharine M. Hardy et al. JOURNAL OF MAMMARY GLAND BIOLOGY AND NEOPLASIA
- TMEPAI, a Transmembrane TGF-β-Inducible Protein, Sequesters Smad Proteins from Active Participation in TGF-β Signaling
- (2010) Yukihide Watanabe et al. MOLECULAR CELL
- Epithelial-mesenchymal transition in cancer development and its clinical significance
- (2009) Masaaki Iwatsuki et al. CANCER SCIENCE
- Targeting Pyk2 for therapeutic intervention
- (2009) Christopher A Lipinski et al. EXPERT OPINION ON THERAPEUTIC TARGETS
- The basics of epithelial-mesenchymal transition
- (2009) Raghu Kalluri et al. JOURNAL OF CLINICAL INVESTIGATION
- Transforming Growth Factor Depletion Is the Primary Determinant of Smad Signaling Kinetics
- (2009) D. C. Clarke et al. MOLECULAR AND CELLULAR BIOLOGY
- Endocytosis and signalling: intertwining molecular networks
- (2009) Alexander Sorkin et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- Met induces mammary tumors with diverse histologies and is associated with poor outcome and human basal breast cancer
- (2009) M. G. Ponzo et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Focal Adhesion Kinase-Related Proline-Rich Tyrosine Kinase 2 and Focal Adhesion Kinase Are Co-Overexpressed in Early-Stage and Invasive ErbB-2-Positive Breast Cancer and Cooperate for Breast Cancer Cell Tumorigenesis and Invasiveness
- (2008) Emy Behmoaram et al. AMERICAN JOURNAL OF PATHOLOGY
- Proline-rich tyrosine kinase 2 (Pyk2) promotes proliferation and invasiveness of hepatocellular carcinoma cells through c-Src/ERK activation
- (2008) C. K. Sun et al. CARCINOGENESIS
- Epithelial-Mesenchymal Transition: At the Crossroads of Development and Tumor Metastasis
- (2008) Jing Yang et al. DEVELOPMENTAL CELL
- Up-regulation of proline-rich tyrosine kinase 2 in non-small cell lung cancer
- (2008) Siyang Zhang et al. LUNG CANCER
- STAT3 inhibition in prostate and pancreatic cancer lines by STAT3 binding sequence oligonucleotides: differential activity between 5′ and 3′ ends
- (2008) H. Dan Lewis et al. MOLECULAR CANCER THERAPEUTICS
- Robust, Tunable Biological Oscillations from Interlinked Positive and Negative Feedback Loops
- (2008) T. Y.-C. Tsai et al. SCIENCE
- Twist is a transcriptional repressor of E-cadherin gene expression in breast cancer
- (2007) Farhad Vesuna et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
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