Augmented Cardiac Hypertrophy in Response to Pressure Overload in Mice Lacking ELTD1

Attenuation of CHOP-mediated Myocardial Apoptosis in Pressure-overloaded Dominant Negative p38α Mitogen-activated Protein Kinase Mice

(2011) Flori R. Sari et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

Regulators of G-Protein Signaling in the Heart and Their Potential as Therapeutic Targets

(2011) Peng Zhang et al.   CIRCULATION RESEARCH

G Protein Coupled Receptor Kinases as Therapeutic Targets in Cardiovascular Disease

(2011) Stephen L. Belmonte et al.   CIRCULATION RESEARCH

Intracellular or extracellular heat shock protein 70 differentially regulates cardiac remodelling in pressure overload mice

(2010) Wen-Feng Cai et al.   CARDIOVASCULAR RESEARCH

Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart

(2010) E. E. Creemers et al.   CARDIOVASCULAR RESEARCH

Effect of pressure overload-induced hypertrophy on the expression and localization of p38 MAP kinase isoforms in the mouse heart

(2010) Dharmendra Dingar et al.   CELLULAR SIGNALLING

Molecular distinction between physiological and pathological cardiac hypertrophy: Experimental findings and therapeutic strategies

(2010) Bianca C. Bernardo et al.   PHARMACOLOGY & THERAPEUTICS

Mitogen-Activated Protein Kinase Signaling in the Heart: Angels Versus Demons in a Heart-Breaking Tale

(2010) Beth A. Rose et al.   PHYSIOLOGICAL REVIEWS

Regulator of G protein signaling 5 protects against cardiac hypertrophy and fibrosis during biomechanical stress of pressure overload

(2010) H. Li et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Connective Tissue Growth Factor Overexpression in Cardiomyocytes Promotes Cardiac Hypertrophy and Protection against Pressure Overload

(2009) Anna N. Panek et al.   PLoS One

A new type of ERK1/2 autophosphorylation causes cardiac hypertrophy

(2008) Kristina Lorenz et al.   NATURE MEDICINE

The Ca2+ ATPase of cardiac sarcoplasmic reticulum: Physiological role and relevance to diseases

(2007) Giuseppe Inesi et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS