Differential response of normal and malignant urothelial cells to CHK1 and ATM inhibitors
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Title
Differential response of normal and malignant urothelial cells to CHK1 and ATM inhibitors
Authors
Keywords
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Journal
ONCOGENE
Volume 34, Issue 22, Pages 2887-2896
Publisher
Springer Nature
Online
2014-07-21
DOI
10.1038/onc.2014.221
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- (2011) K. A. Cole et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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