Early Down-Regulation of PKCδ as a Pro-Survival Mechanism in Huntington’s Disease
Published 2013 View Full Article
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Title
Early Down-Regulation of PKCδ as a Pro-Survival Mechanism in Huntington’s Disease
Authors
Keywords
Cell death, Huntingtin, Lamin B, PKCδ dominant negative
Journal
NEUROMOLECULAR MEDICINE
Volume 16, Issue 1, Pages 25-37
Publisher
Springer Nature
Online
2013-07-29
DOI
10.1007/s12017-013-8248-8
References
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Related references
Note: Only part of the references are listed.- Activation of Elk-1 participates as a neuroprotective compensatory mechanism in models of Huntington’s disease
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- Huntington's Disease: Can Mice Lead the Way to Treatment?
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- Continuous and Periodic Expansion of CAG Repeats in Huntington's Disease R6/1 Mice
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- (2009) Jose R Pineda et al. Molecular Brain
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- CAG repeat lengths ≥335 attenuate the phenotype in the R6/2 Huntington's disease transgenic mouse
- (2008) I. Dragatsis et al. NEUROBIOLOGY OF DISEASE
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- (2008) John Brognard et al. TRENDS IN ENDOCRINOLOGY AND METABOLISM
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