Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease
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Title
Increased PKA signaling disrupts recognition memory and spatial memory: role in Huntington's disease
Authors
Keywords
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Journal
HUMAN MOLECULAR GENETICS
Volume 20, Issue 21, Pages 4232-4247
Publisher
Oxford University Press (OUP)
Online
2011-08-12
DOI
10.1093/hmg/ddr351
References
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- (2009) D. A. Simmons et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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- Constitutive activation of the G-protein subunit G s within forebrain neurons causes PKA-dependent alterations in fear conditioning and cortical Arc mRNA expression
- (2008) M. P. Kelly et al. LEARNING & MEMORY
- Brain-derived neurotrophic factor modulates the severity of cognitive alterations induced by mutant huntingtin: Involvement of phospholipaseCγ activity and glutamate receptor expression
- (2008) A. Giralt et al. NEUROSCIENCE
- Protein kinase A inhibits a consolidated form of memory in Drosophila
- (2008) J. Horiuchi et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Microtubule-associated protein tau in development, degeneration and protection of neurons
- (2008) Jian-Zhi Wang et al. PROGRESS IN NEUROBIOLOGY
- Selective phosphodiesterase inhibitors: a promising target for cognition enhancement
- (2008) Olga A. H. Reneerkens et al. PSYCHOPHARMACOLOGY
- Gene–environment interactions modulating cognitive function and molecular correlates of synaptic plasticity in Huntington’s disease transgenic mice
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- Disruption of striatal glutamatergic transmission induced by mutant huntingtin involves remodeling of both postsynaptic density and NMDA receptor signaling
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