Chrysin attenuates interstitial fibrosis and improves cardiac function in a rat model of acute myocardial infarction
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Title
Chrysin attenuates interstitial fibrosis and improves cardiac function in a rat model of acute myocardial infarction
Authors
Keywords
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Journal
JOURNAL OF MOLECULAR HISTOLOGY
Volume -, Issue -, Pages -
Publisher
Springer Nature America, Inc
Online
2018-09-17
DOI
10.1007/s10735-018-9793-0
References
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Related references
Note: Only part of the references are listed.- Cardiac fibrosis in myocardial infarction—from repair and remodeling to regeneration
- (2016) Virpi Talman et al. CELL AND TISSUE RESEARCH
- Chrysin, a PPAR-γ agonist improves myocardial injury in diabetic rats through inhibiting AGE-RAGE mediated oxidative stress and inflammation
- (2016) Neha Rani et al. CHEMICO-BIOLOGICAL INTERACTIONS
- Matrix metalloproteinases as input and output signals for post-myocardial infarction remodeling
- (2016) Merry L. Lindsey et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Chrysin attenuates liver fibrosis and hepatic stellate cell activation through TGF-β/Smad signaling pathway
- (2015) Cornel Balta et al. CHEMICO-BIOLOGICAL INTERACTIONS
- Osteopontin is indispensible for AP1-mediated angiotensin II-related miR-21 transcription during cardiac fibrosis
- (2015) Johan M. Lorenzen et al. EUROPEAN HEART JOURNAL
- Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction
- (2015) Andrew P. Voorhees et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Chrysin inhibits diabetic renal tubulointerstitial fibrosis through blocking epithelial to mesenchymal transition
- (2015) Min-Kyung Kang et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- Inhibition of TGF-β by a novel PPAR-γ agonist, chrysin, salvages β-receptor stimulated myocardial injury in rats through MAPKs-dependent mechanism
- (2015) Neha Rani et al. Nutrition & Metabolism
- P442Chrysin alleviates isoproterenol-induced myocardial infarction in diabetic rats through modulation of PPAR-gamma
- (2014) N Rani et al. CARDIOVASCULAR RESEARCH
- Chrysin alleviates acute doxorubicin cardiotoxicity in rats via suppression of oxidative stress, inflammation and apoptosis
- (2014) Eman M. Mantawy et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- PPAR-γ activation by rosiglitazone suppresses angiotensin II-mediated proliferation and phenotypictransition in cardiac fibroblasts via inhibition of activation of activator protein 1
- (2013) Xiaoyang Hou et al. EUROPEAN JOURNAL OF PHARMACOLOGY
- Fibroblasts in myocardial infarction: A role in inflammation and repair
- (2013) Arti V. Shinde et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
- Beneficial effects of Chrysin against Methotrexate-induced hepatotoxicity via attenuation of oxidative stress and apoptosis
- (2013) Nemat Ali et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Telmisartan, a unique ARB, improves left ventricular remodeling of infarcted heart by activating PPAR gamma
- (2011) Yasuhiro Maejima et al. LABORATORY INVESTIGATION
- Proteomic analysis identifies in vivo candidate matrix metalloproteinase-9 substrates in the left ventricle post-myocardial infarction
- (2010) Rogelio Zamilpa et al. PROTEOMICS
- Pioglitazone Improves Cardiac Function and Alters Myocardial Substrate Metabolism Without Affecting Cardiac Triglyceride Accumulation and High-Energy Phosphate Metabolism in Patients With Well-Controlled Type 2 Diabetes Mellitus
- (2009) Rutger W. van der Meer et al. CIRCULATION
- Extracellular matrix fibrotic markers in heart failure
- (2009) Faiez Zannad et al. HEART FAILURE REVIEWS
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