p87 and p101 Subunits Are Distinct Regulators Determining Class IBPhosphoinositide 3-Kinase (PI3K) Specificity
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Title
p87 and p101 Subunits Are Distinct Regulators Determining Class IBPhosphoinositide 3-Kinase (PI3K) Specificity
Authors
Keywords
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Journal
JOURNAL OF BIOLOGICAL CHEMISTRY
Volume 288, Issue 43, Pages 31059-31068
Publisher
American Society for Biochemistry & Molecular Biology (ASBMB)
Online
2013-09-07
DOI
10.1074/jbc.m113.508234
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- (2012) Bart Vanhaesebroeck et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
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- (2011) Michael C. Schmid et al. CANCER CELL
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- (2011) Sze Ki Luk et al. EUROPEAN JOURNAL OF CANCER
- Structure of Lipid Kinase p110β/p85β Elucidates an Unusual SH2-Domain-Mediated Inhibitory Mechanism
- (2011) Xuxiao Zhang et al. MOLECULAR CELL
- Integrating Cardiac PIP3 and cAMP Signaling through a PKA Anchoring Function of p110γ
- (2011) Alessia Perino et al. MOLECULAR CELL
- Differential roles for the p101 and p84 regulatory subunits of PI3Kγ in tumor growth and metastasis
- (2011) J A Brazzatti et al. ONCOGENE
- Structural Basis for Activation and Inhibition of Class I Phosphoinositide 3-Kinases
- (2011) Oscar Vadas et al. Science Signaling
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- (2010) Federico Damilano et al. Annals of the New York Academy of Sciences
- Hot-spot mutations in p110α of phosphatidylinositol 3-kinase (PI3K): Differential interactions with the regulatory subunit p85 and with RAS
- (2010) Li Zhao et al. CELL CYCLE
- The emerging mechanisms of isoform-specific PI3K signalling
- (2010) Bart Vanhaesebroeck et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- A biochemical mechanism for the oncogenic potential of the p110 catalytic subunit of phosphoinositide 3-kinase
- (2010) H. A. Dbouk et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Form and flexibility in phosphoinositide 3-kinases
- (2009) Roger Williams et al. BIOCHEMICAL SOCIETY TRANSACTIONS
- Fine tuning the immune response with PI3K
- (2009) David A. Fruman et al. IMMUNOLOGICAL REVIEWS
- Ras is an indispensable coregulator of the class IB phosphoinositide 3-kinase p87/p110
- (2009) B. Kurig et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Gβγ-Copurified Lipid Kinase Impurity from Sf9 Cells
- (2009) Aliaksei Shymanets et al. PROTEIN AND PEPTIDE LETTERS
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- Pasteurella multocida toxin activates Gβγ dimers of heterotrimeric G proteins
- (2008) Inga Preuß et al. CELLULAR SIGNALLING
- The p110 isoform of phosphoinositide 3-kinase signals downstream of G protein-coupled receptors and is functionally redundant with p110
- (2008) J. Guillermet-Guibert et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Phosphoinositide 3-Kinase p110 Activity: Key Role in Metabolism and Mammary Gland Cancer but Not Development
- (2008) E. Ciraolo et al. Science Signaling
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