标题
Matrix remodeling in systemic sclerosis
作者
关键词
Scleroderma, Scleroderma Patient, Early Growth Response Gene, Dermal Fibrosis, Scleroderma Skin
出版物
Seminars in Immunopathology
Volume 37, Issue 5, Pages 559-563
出版商
Springer Nature
发表日期
2015-07-03
DOI
10.1007/s00281-015-0508-2
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Fibrosis, Vascular Activation, and Immune Abnormalities Resembling Systemic Sclerosis in Bleomycin-Treated Fli-1-Haploinsufficient Mice
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- A Role of Myocardin Related Transcription Factor-A (MRTF-A) in Scleroderma Related Fibrosis
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- Failed Degradation of JunB Contributes to Overproduction of Type I Collagen and Development of Dermal Fibrosis in Patients With Systemic Sclerosis
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- Endoglin promotes TGF-β/Smad1 signaling in scleroderma fibroblasts
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- Loss of PTEN Expression by Dermal Fibroblasts Causes Skin Fibrosis
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- Egr-1 Induces a Profibrotic Injury/Repair Gene Program Associated with Systemic Sclerosis
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- Modulation of collagen type I, fibronectin and dermal fibroblast function and activity, in systemic sclerosis by the antioxidant epigallocatechin-3-gallate
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- Essential Roles for Early Growth Response Transcription Factor Egr-1 in Tissue Fibrosis and Wound Healing
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- JunB mediates enhancer/promoter activity of COL1A2 following TGF-β induction
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- Tissue stiffness, latent TGF-β1 Activation, and mechanical signal transduction: Implications for the pathogenesis and treatment of fibrosis
- (2009) Boris Hinz Current Rheumatology Reports
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