Poor maternal nutrition leads to alterations in oxidative stress, antioxidant defense capacity, and markers of fibrosis in rat islets: potential underlying mechanisms for development of the diabetic phenotype in later life

Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell

(2009) P. Newsholme et al.   DIABETOLOGIA

Poor maternal nutrition followed by accelerated postnatal growth leads to telomere shortening and increased markers of cell senescence in rat islets

(2009) J. L. Tarry-Adkins et al.   FASEB JOURNAL

Lipoperoxidation and antioxidant capacity in patients with poorly controlled type 2 diabetes

(2009) M Lodovici et al.   TOXICOLOGY AND INDUSTRIAL HEALTH

NOX family NADPH oxidases in liver and in pancreatic islets: a role in the metabolic syndrome and diabetes?

(2008) Cécile Guichard et al.   BIOCHEMICAL SOCIETY TRANSACTIONS

Molecular insights into connective tissue growth factor action in rat pancreatic stellate cells

(2008) Anna Karger et al.   CELLULAR SIGNALLING

AT1-receptor blockade by telmisartan upregulates GTP-cyclohydrolase I and protects eNOS in diabetic rats

(2008) Philip Wenzel et al.   FREE RADICAL BIOLOGY AND MEDICINE

Increased Serum Levels of Ne-(hexanoyl)lysine, A New Marker of Oxidative Stress, in Systemic Sclerosis

(2008) Kazuhiro Shimizu et al.   JOURNAL OF RHEUMATOLOGY

NADPH oxidase plays a crucial role in the activation of pancreatic stellate cells

(2007) Atsushi Masamune et al.   AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY