Increased expression of claudin-17 promotes a malignant phenotype in hepatocyte via Tyk2/Stat3 signaling and is associated with poor prognosis in patients with hepatocellular carcinoma
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Title
Increased expression of claudin-17 promotes a malignant phenotype in hepatocyte via Tyk2/Stat3 signaling and is associated with poor prognosis in patients with hepatocellular carcinoma
Authors
Keywords
Hepatocytes, Tight junction, Migration, CLDN17, Tyrosine kinase 2, Signal transducer and activator of transcription 3
Journal
Diagnostic Pathology
Volume 13, Issue 1, Pages -
Publisher
Springer Nature America, Inc
Online
2018-09-16
DOI
10.1186/s13000-018-0749-1
References
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- Claudin-1 induces epithelial–mesenchymal transition through activation of the c-Abl-ERK signaling pathway in human liver cells
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- Claudin-6, 7, or 9 Overexpression in the Human Gastric Adenocarcinoma Cell Line AGS Increases Its Invasiveness, Migration, and Proliferation Rate
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- The Claudin family and its role in cancer and metastasis
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- Crosstalk of tight junction components with signaling pathways
- (2007) Lorenza González-Mariscal et al. BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES
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