Translationally controlled tumour protein is associated with podocyte hypertrophy in a mouse model of type 1 diabetes
Published 2012 View Full Article
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Title
Translationally controlled tumour protein is associated with podocyte hypertrophy in a mouse model of type 1 diabetes
Authors
Keywords
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Journal
DIABETOLOGIA
Volume 55, Issue 4, Pages 1205-1217
Publisher
Springer Nature
Online
2012-02-04
DOI
10.1007/s00125-012-2467-7
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Note: Only part of the references are listed.- Role of mTOR in podocyte function and diabetic nephropathy in humans and mice
- (2011) Markus Gödel et al. JOURNAL OF CLINICAL INVESTIGATION
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- (2010) Gavin I. Welsh et al. Cell Metabolism
- BTBR Ob/Ob Mutant Mice Model Progressive Diabetic Nephropathy
- (2010) K. L. Hudkins et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
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- Molecular Basis of the Acceleration of the GDP-GTP Exchange of Human Ras Homolog Enriched in Brain by Human Translationally Controlled Tumor Protein
- (2009) Xianchi Dong et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- mTOR signaling at a glance
- (2009) M. Laplante et al. JOURNAL OF CELL SCIENCE
- PKC- 1 Mediates Glucose-Induced Akt Activation and TGF- 1 Upregulation in Mesangial Cells
- (2009) D. Wu et al. JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY
- TCTP protects from apoptotic cell death by antagonizing bax function
- (2008) L Susini et al. CELL DEATH AND DIFFERENTIATION
- Lentiviral gene transfer to reduce atherosclerosis progression by long-term CC-chemokine inhibition
- (2008) C A Bursill et al. GENE THERAPY
- Failure to phosphorylate AKT in podocytes from mice with early diabetic nephropathy promotes cell death
- (2008) T. Tejada et al. KIDNEY INTERNATIONAL
- mTOR-Raptor Binds and Activates SGK1 to Regulate p27 Phosphorylation
- (2008) Feng Hong et al. MOLECULAR CELL
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