Shear stress is a positive regulator of thimet oligopeptidase (EC3.4.24.15) in vascular endothelial cells: consequences for MHC1 levels

Tight control – decision-making during T cell–vascular endothelial cell interaction

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Arterial flow reduces oxidative stress via an antioxidant response element and Oct-1 binding site within the NADPH oxidase 4 promoter in endothelial cells

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Stabilization of brain microvascular endothelial barrier function by shear stress involves VE-cadherin signaling leading to modulation of pTyr-occludin levels

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Vasoprotective and Atheroprotective Effects of Angiotensin (1-7) in Apolipoprotein E–Deficient Mice

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ACE2: more of Ang-(1–7) or less Ang II?

(2010) Carlos M Ferrario   CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION

Antigen processing by nardilysin and thimet oligopeptidase generates cytotoxic T cell epitopes

(2010) Jan H Kessler et al.   NATURE IMMUNOLOGY

Interaction with calmodulin is important for the secretion of thimet oligopeptidase following stimulation

(2009) Lilian C. Russo et al.   FEBS Journal

Down-regulation of neprilysin (EC3.4.24.11) expression in vascular endothelial cells by laminar shear stress involves NADPH oxidase-dependent ROS production

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A Biological Role for the Gonadotrophin-Releasing Hormone (GnRH) Metabolite, GnRH-(1-5)

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Mild inflammatory activation of mammary arteries in patients with acute coronary syndromes

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NADPH oxidase has a directional response to shear stress

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Mechanotransduction in vascular physiology and atherogenesis

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