NADPH oxidase activation by hyperglycaemia in cardiomyocytes is independent of glucose metabolism but requires SGLT1

Biology of Human Sodium Glucose Transporters

(2011) Ernest M. Wright et al.   PHYSIOLOGICAL REVIEWS

Superoxide production by NAD(P)H oxidase and mitochondria is increased in genetically obese and hyperglycemic rat heart and aorta before the development of cardiac dysfunction. The role of glucose-6-phosphate dehydrogenase-derived NADPH

(2009) Sabrina Serpillon et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Calpain activation contributes to hyperglycaemia-induced apoptosis in cardiomyocytes

(2009) Y. Li et al.   CARDIOVASCULAR RESEARCH

SGLT1 is a novel cardiac glucose transporter that is perturbed in disease states

(2009) S. K. Banerjee et al.   CARDIOVASCULAR RESEARCH

Rac1 Is Required for Cardiomyocyte Apoptosis During Hyperglycemia

(2009) E. Shen et al.   DIABETES

Synergistic activation of glucose-6-phosphate dehydrogenase and NAD(P)H oxidase by Src kinase elevates superoxide in type 2 diabetic, Zucker fa/fa, rat liver

(2009) Rakhee S. Gupte et al.   FREE RADICAL BIOLOGY AND MEDICINE

NADPH oxidase signaling and cardiac myocyte function

(2009) Ashwin Akki et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

O-GlcNAc modification of FoxO1 increases its transcriptional activity: A role in the glucotoxicity phenomenon?

(2008) MeiShiue Kuo et al.   BIOCHIMIE

Unique Hexosaminidase Reduces Metabolic Survival Signal and Sensitizes Cardiac Myocytes to Hypoxia/Reoxygenation Injury

(2008) Gladys A. Ngoh et al.   CIRCULATION RESEARCH