Co-expression of NaVβ subunits alters the kinetics of inhibition of voltage-gated sodium channels by pore-blocking μ-conotoxins

Pharmacological fractionation of tetrodotoxin-sensitive sodium currents in rat dorsal root ganglion neurons by μ-conotoxins

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(2012) Stephen G. Waxman   JOURNAL OF PHYSIOLOGY-LONDON

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(2012) William A. Catterall   JOURNAL OF PHYSIOLOGY-LONDON

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NaV  Subunits Modulate the Inhibition of NaV1.8 by the Analgesic Gating Modifier  O-Conotoxin MrVIB

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 -Conotoxins that differentially block sodium channels NaV1.1 through 1.8 identify those responsible for action potentials in sciatic nerve

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Regulatory role of voltage-gated Na+ channel β subunits in sensory neurons

(2011) Mohamed Chahine   Frontiers in Pharmacology

μ-Conotoxin KIIIA Derivatives with Divergent Affinities versus Efficacies in Blocking Voltage-Gated Sodium Channels

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Unexpected synergism between Tetrodotoxin and μ-conotoxin in blocking voltage-gated sodium channels

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Cooccupancy of the Outer Vestibule of Voltage-Gated Sodium Channels by μ-Conotoxin KIIIA and Saxitoxin or Tetrodotoxin

(2010) Min-Min Zhang et al.   JOURNAL OF NEUROPHYSIOLOGY

Differential Evolution of Voltage-Gated Sodium Channels in Tetrapods and Teleost Fishes

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Electrophysiology and beyond: Multiple roles of Na+ channel β subunits in development and disease

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Control of transient, resurgent, and persistent current by open-channel block by Na channel  4 in cultured cerebellar granule neurons

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Voltage-Gated Sodium Channels: Therapeutic Targets for Pain

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Specificity, affinity and efficacy of iota-conotoxin RXIA, an agonist of voltage-gated sodium channels NaV1.2, 1.6 and 1.7

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Comparative study of the distribution of the α-subunits of voltage-gated sodium channels in normal and axotomized rat dorsal root ganglion neurons

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