Aldara®-induced skin inflammation: studies of patients with psoriasis
Published 2014 View Full Article
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Title
Aldara®-induced skin inflammation: studies of patients with psoriasis
Authors
Keywords
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Journal
BRITISH JOURNAL OF DERMATOLOGY
Volume 172, Issue 2, Pages 345-353
Publisher
Wiley
Online
2014-07-02
DOI
10.1111/bjd.13236
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Note: Only part of the references are listed.- IL-1R1 Signaling Facilitates Munro’s Microabscess Formation in Psoriasiform Imiquimod-Induced Skin Inflammation
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- Aldara activates TLR7-independent immune defence
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- Rorγt+ innate lymphocytes and γδ T cells initiate psoriasiform plaque formation in mice
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- Caspase-14-Deficient Mice Are More Prone to the Development of Parakeratosis
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- IL-17C regulates the innate immune function of epithelial cells in an autocrine manner
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- Mutual Antagonism of T Cells Causing Psoriasis and Atopic Eczema
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- Genome-Wide Expression Profiling of Five Mouse Models Identifies Similarities and Differences with Human Psoriasis
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- Preferential inhibition of the mRNA expression of p38 mitogen-activated protein kinase regulated cytokines in psoriatic skin by anti-TNFα therapy
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- New Insights in the Immunologic Basis of Psoriasis
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- Imiquimod-Induced Psoriasis-Like Skin Inflammation in Mice Is Mediated via the IL-23/IL-17 Axis
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- Production of interleukin 22 but not interleukin 17 by a subset of human skin-homing memory T cells
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- T-Helper 17 Cells in Psoriatic Plaques and Additional Genetic Links between IL-23 and Psoriasis
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- Resident and “Inflammatory” Dendritic Cells in Human Skin
- (2008) Lisa C. Zaba et al. JOURNAL OF INVESTIGATIVE DERMATOLOGY
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