PAK4 promotes kinase-independent stabilization of RhoU to modulate cell adhesion
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
PAK4 promotes kinase-independent stabilization of RhoU to modulate cell adhesion
Authors
Keywords
-
Journal
JOURNAL OF CELL BIOLOGY
Volume 211, Issue 4, Pages 863-879
Publisher
Rockefeller University Press
Online
2015-11-24
DOI
10.1083/jcb.201501072
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- The atypical Rho GTPase, RhoU, regulates cell-adhesion molecules during cardiac morphogenesis
- (2014) Michael Dickover et al. DEVELOPMENTAL BIOLOGY
- RhoJ interacts with the GIT-PIX complex and regulates focal adhesion disassembly
- (2014) E. Wilson et al. JOURNAL OF CELL SCIENCE
- NMR binding and crystal structure reveal that intrinsically-unstructured regulatory domain auto-inhibits PAK4 by a mechanism different from that of PAK1
- (2013) Wei Wang et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- Ubiquitin — omics reveals novel networks and associations with human disease
- (2013) Benedikt M Kessler CURRENT OPINION IN CHEMICAL BIOLOGY
- P21-activated kinase 4 – Not just one of the PAK
- (2013) Anna E. Dart et al. EUROPEAN JOURNAL OF CELL BIOLOGY
- Cullin 5 destabilizes Cas to inhibit Src-dependent cell transformation
- (2013) A. Teckchandani et al. JOURNAL OF CELL SCIENCE
- Pak6 protein kinase is a novel effector of an atypical Rho family GTPase Chp/RhoV
- (2012) M. V. Shepelev et al. BIOCHEMISTRY-MOSCOW
- Group I and II mammalian PAKs have different modes of activation by Cdc42
- (2012) Yohendran Baskaran et al. EMBO REPORTS
- Proteomic Analyses Reveal Divergent Ubiquitylation Site Patterns in Murine Tissues
- (2012) Sebastian A. Wagner et al. MOLECULAR & CELLULAR PROTEOMICS
- Knockdown of PAK4 or PAK1 Inhibits the Proliferation of Mutant KRAS Colon Cancer Cells Independently of RAF/MEK/ERK and PI3K/AKT Signaling
- (2012) H. Tabusa et al. MOLECULAR CANCER RESEARCH
- ErbB2-Driven Breast Cancer Cell Invasion Depends on a Complex Signaling Network Activating Myeloid Zinc Finger-1-Dependent Cathepsin B Expression
- (2012) Bo Rafn et al. MOLECULAR CELL
- Coordination between RAB GTPase and phosphoinositide regulation and functions
- (2012) Steve Jean et al. NATURE REVIEWS MOLECULAR CELL BIOLOGY
- PAK4 kinase activity and somatic mutation promote carcinoma cell motility and influence inhibitor sensitivity
- (2012) A D Whale et al. ONCOGENE
- p21-Activated kinase 4 promotes prostate cancer progression through CREB
- (2012) M-H Park et al. ONCOGENE
- Analysis of Rho GTPase expression in T-ALL identifies RhoU as a target for Notch involved in T-ALL cell migration
- (2012) P J Bhavsar et al. ONCOGENE
- Type II p21-activated kinases (PAKs) are regulated by an autoinhibitory pseudosubstrate
- (2012) B. H. Ha et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- K63-Linked Ubiquitination in Kinase Activation and Cancer
- (2012) Guocan Wang et al. Frontiers in Oncology
- Nucleo-cytoplasmic shuttling of PAK4 modulates β-catenin intracellular translocation and signaling
- (2011) Yan Li et al. BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH
- LCH-7749944, a novel and potent p21-activated kinase 4 inhibitor, suppresses proliferation and invasion in human gastric cancer cells
- (2011) Jian Zhang et al. CANCER LETTERS
- Notice of Retraction
- (2011) CARCINOGENESIS
- Structure and function of focal adhesions
- (2011) Bernhard Wehrle-Haller CURRENT OPINION IN CELL BIOLOGY
- SHARPIN is an endogenous inhibitor of β1-integrin activation
- (2011) Juha K. Rantala et al. NATURE CELL BIOLOGY
- Subgroup II PAK-mediated phosphorylation regulates Ran activity during mitosis
- (2010) Guillaume Bompard et al. JOURNAL OF CELL BIOLOGY
- The role of ubiquitylation and degradation in RhoGTPase signalling
- (2010) Micha Nethe et al. JOURNAL OF CELL SCIENCE
- PAK4: a pluripotent kinase that regulates prostate cancer cell adhesion
- (2010) C. M. Wells et al. JOURNAL OF CELL SCIENCE
- Focal-adhesion targeting links caveolin-1 to a Rac1-degradation pathway
- (2010) M. Nethe et al. JOURNAL OF CELL SCIENCE
- Cdc42 Regulates Apical Junction Formation in Human Bronchial Epithelial Cells through PAK4 and Par6B
- (2010) Sean W. Wallace et al. MOLECULAR BIOLOGY OF THE CELL
- The protein kinase Pak4 disrupts mammary acinar architecture and promotes mammary tumorigenesis
- (2010) Y Liu et al. ONCOGENE
- p21-activated kinase 4 regulates ovarian cancer cell proliferation, migration, and invasion and contributes to poor prognosis in patients
- (2010) M. K. Y. Siu et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Small-molecule p21-activated kinase inhibitor PF-3758309 is a potent inhibitor of oncogenic signaling and tumor growth
- (2010) B. W. Murray et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- The RhoU/Wrch1 Rho GTPase gene is a common transcriptional target of both the gp130/STAT3 and Wnt-1 pathways
- (2009) Davide Schiavone et al. BIOCHEMICAL JOURNAL
- Chromosomal changes in aggressive breast cancers with basal-like features
- (2009) Wayne Yu et al. CANCER GENETICS AND CYTOGENETICS
- Paxillin nuclear-cytoplasmic localization is regulated by phosphorylation of the LD4motif: evidence that nuclear paxillin promotes cell proliferation
- (2008) Jing-Ming Dong et al. BIOCHEMICAL JOURNAL
- A PAK4–LIMK1 pathway drives prostate cancer cell migration downstream of HGF
- (2008) Tasneem Ahmed et al. CELLULAR SIGNALLING
- The Pak4 Protein Kinase Plays a Key Role in Cell Survival and Tumorigenesis in Athymic Mice
- (2008) Y. Liu et al. MOLECULAR CANCER RESEARCH
- UnPAKing the class differences among p21-activated kinases
- (2008) Jeyanthy Eswaran et al. TRENDS IN BIOCHEMICAL SCIENCES
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationPublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More