Histone Acetyltransferase GCN5 Regulates Osteogenic Differentiation of Mesenchymal Stem Cells by Inhibiting NF-κB
Published 2015 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Histone Acetyltransferase GCN5 Regulates Osteogenic Differentiation of Mesenchymal Stem Cells by Inhibiting NF-κB
Authors
Keywords
-
Journal
JOURNAL OF BONE AND MINERAL RESEARCH
Volume 31, Issue 2, Pages 391-402
Publisher
Wiley
Online
2015-09-07
DOI
10.1002/jbmr.2704
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Gremlin 1 Identifies a Skeletal Stem Cell with Bone, Cartilage, and Reticular Stromal Potential
- (2015) Daniel L. Worthley et al. CELL
- Identification and Specification of the Mouse Skeletal Stem Cell
- (2015) Charles K.F. Chan et al. CELL
- Gcn5 and PCAF negatively regulate interferon- production through HAT-independent inhibition of TBK1
- (2014) Q. Jin et al. EMBO REPORTS
- NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB
- (2014) Hengwei Zhang et al. JOURNAL OF CLINICAL INVESTIGATION
- Endothelial Notch activity promotes angiogenesis and osteogenesis in bone
- (2014) Saravana K. Ramasamy et al. NATURE
- Tumor suppressor p53 cooperates with SIRT6 to regulate gluconeogenesis by promoting FoxO1 nuclear exclusion
- (2014) P. Zhang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Flow Cytometric Cell Sorting and In Vitro Pre-Osteoinduction Are Not Requirements for In Vivo Bone Formation by Human Adipose-Derived Stromal Cells
- (2013) Yunsong Liu et al. PLoS One
- NF-κB inhibits osteogenic differentiation of mesenchymal stem cells by promoting β-catenin degradation
- (2013) Jia Chang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- DNA damage drives accelerated bone aging via an NF-κB-dependent mechanism
- (2012) Qian Chen et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Inhibition of Osteogenic Differentiation of Human Adipose-Derived Stromal Cells by Retinoblastoma Binding Protein 2 Repression of RUNX2-Activated Transcription
- (2011) Wenshu Ge et al. STEM CELLS
- Role of NF-κB in the skeleton
- (2010) Deborah Veis Novack CELL RESEARCH
- Proper Gcn5 histone acetyltransferase expression is required for normal anteroposterior patterning of the mouse skeleton
- (2010) Wenchu Lin et al. DEVELOPMENT GROWTH & DIFFERENTIATION
- Gcn5 regulates the dissociation of SWI/SNF from chromatin by acetylation of Swi2/Snf2
- (2010) J.-H. Kim et al. GENES & DEVELOPMENT
- Guidelines for assessment of bone microstructure in rodents using micro-computed tomography
- (2010) Mary L Bouxsein et al. JOURNAL OF BONE AND MINERAL RESEARCH
- Mesenchymal and haematopoietic stem cells form a unique bone marrow niche
- (2010) Simón Méndez-Ferrer et al. NATURE
- Multiple functions of Osterix are required for bone growth and homeostasis in postnatal mice
- (2010) X. Zhou et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- GCN5 is a required cofactor for a ubiquitin ligase that targets NF- B/RelA
- (2009) X. Mao et al. GENES & DEVELOPMENT
- Inhibition of osteoblastic bone formation by nuclear factor-κB
- (2009) Jia Chang et al. NATURE MEDICINE
- Hedgehog Signaling in Mature Osteoblasts Regulates Bone Formation and Resorption by Controlling PTHrP and RANKL Expression
- (2008) Kinglun Kingston Mak et al. DEVELOPMENTAL CELL
- Fibroblast Growth Factor Signaling Uses Multiple Mechanisms To Inhibit Wnt-Induced Transcription in Osteoblasts
- (2008) D. Ambrosetti et al. MOLECULAR AND CELLULAR BIOLOGY
- NF-κB links innate immunity to the hypoxic response through transcriptional regulation of HIF-1α
- (2008) Jordi Rius et al. NATURE
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExplorePublish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn More