Functional analysis of miR-101-3p and Rap1b involved in hepatitis B virus-related hepatocellular carcinoma pathogenesis
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Title
Functional analysis of miR-101-3p and Rap1b involved in hepatitis B virus-related hepatocellular carcinoma pathogenesis
Authors
Keywords
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Journal
Biochemistry and Cell Biology
Volume 92, Issue 2, Pages 152-162
Publisher
Canadian Science Publishing
Online
2014-03-10
DOI
10.1139/bcb-2013-0128
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Note: Only part of the references are listed.- The silence of MUC2 mRNA induced by promoter hypermethylation associated with HBV in Hepatocellular Carcinoma
- (2013) Yang Ling et al. BMC Medical Genetics
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- (2013) Rui-Yan Li et al. CNS Neuroscience & Therapeutics
- Hepatitis B virus X protein represses miRNA-148a to enhance tumorigenesis
- (2013) Xiaojie Xu et al. JOURNAL OF CLINICAL INVESTIGATION
- MicroRNA-143 inhibits cell migration and invasion by targeting matrix metalloproteinase 13 in prostate cancer
- (2013) DEYAO WU et al. Molecular Medicine Reports
- MiR-26a Inhibits Proliferation and Migration of Breast Cancer through Repression of MCL-1
- (2013) Jie Gao et al. PLoS One
- Downregulation of miR-101 in gastric cancer correlates with cyclooxygenase-2 overexpression and tumor growth
- (2012) Xiao-Pu He et al. FEBS Journal
- miR-518b is down-regulated, and involved in cell proliferation and invasion by targeting Rap1b in esophageal squamous cell carcinoma
- (2012) Mingxin Zhang et al. FEBS LETTERS
- Oncofetal antigen glypican-3 as a promising early diagnostic marker for hepatocellular carcinoma
- (2012) Min Yao et al. Hepatobiliary & Pancreatic Diseases International
- Regulation of RAP1B by miR-139 suppresses human colorectal carcinoma cell proliferation
- (2012) Haiyan Guo et al. INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY
- miR-338-3p Is Down-Regulated by Hepatitis B Virus X and Inhibits Cell Proliferation by Targeting the 3′-UTR Region of CyclinD1
- (2012) Xiaoyu Fu et al. INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
- MiR-101 Is Involved in Human Breast Carcinogenesis by Targeting Stathmin1
- (2012) Rui Wang et al. PLoS One
- Circulating microRNAs in hepatitis B virus-infected patients
- (2011) F. Ji et al. JOURNAL OF VIRAL HEPATITIS
- Hepatitis B virus and hepatocellular carcinoma at the miRNA level
- (2011) Zhen-Zhen Zhang WORLD JOURNAL OF GASTROENTEROLOGY
- MicroRNAs in Hepatobiliary and Pancreatic Cancers
- (2011) Yoshimasa Saito et al. Frontiers in Genetics
- MicroRNA-602 regulating tumor suppressive gene RASSF1A is over-expressed in hepatitis B virus-infected liver and hepatocellular carcinoma
- (2010) Lian Yang et al. CANCER BIOLOGY & THERAPY
- Down-regulated microRNA-152 induces aberrant DNA methylation in hepatitis B virus-related hepatocellular carcinoma by targeting DNA methyltransferase 1
- (2010) Jinfeng Huang et al. HEPATOLOGY
- Epidemiology of chronic hepatitis B virus infection, hepatocellular carcinoma, and hepatitis B virus-induced hepatocellular carcinoma
- (2010) M.C. Kew PATHOLOGIE BIOLOGIE
- New Tricks for Animal MicroRNAs: Targeting of Amino Acid Coding Regions at Conserved and Nonconserved Sites
- (2009) I. Rigoutsos CANCER RESEARCH
- MicroRNA-101, Down-regulated in Hepatocellular Carcinoma, Promotes Apoptosis and Suppresses Tumorigenicity
- (2009) H. Su et al. CANCER RESEARCH
- Up-regulated microRNA-143 transcribed by nuclear factor kappa B enhances hepatocarcinoma metastasis by repressing fibronectin expression
- (2009) Xiaoying Zhang et al. HEPATOLOGY
- Association of MicroRNA Expression in Hepatocellular Carcinomas with Hepatitis Infection, Cirrhosis, and Patient Survival
- (2008) J. Jiang et al. CLINICAL CANCER RESEARCH
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