Identification of Novel 14-3-3 Residues That Are Critical for Isoform-specific Interaction with GluN2C to RegulateN-Methyl-d-aspartate (NMDA) Receptor Trafficking
Identification of Novel 14-3-3 Residues That Are Critical for Isoform-specific Interaction with GluN2C to RegulateN-Methyl-d-aspartate (NMDA) Receptor Trafficking
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Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
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