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Molecular Mechanisms Leading to Neuroprotection/Ischemic Tolerance: Effect of Preconditioning on the Stress Reaction of Endoplasmic Reticulum

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CALRETICULIN DOWNREGULATION IS ASSOCIATED WITH FGF-2-INDUCED ANGIOGENESIS THROUGH CALCINEURIN PATHWAY IN ISCHEMIC MYOCARDIUM

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Acidic pre-conditioning suppresses apoptosis and increases expression of Bcl-xL in coronary endothelial cells under simulated ischaemia

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ISCHEMIC POSTCONDITIONING PROTECTS MYOCARDIUM FROM ISCHEMIA/REPERFUSION INJURY THROUGH ATTENUATING ENDOPLASMIC RETICULUM STRESS

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