Resveratrol promotes endothelial cell wound healing under laminar shear stress through an estrogen receptor-α-dependent pathway

Estrogen, vascular estrogen receptor and hormone therapy in postmenopausal vascular disease

(2013) Raouf A. Khalil   BIOCHEMICAL PHARMACOLOGY

Altered nitric oxide production mediates matrix-specific PAK2 and NF-κB activation by flow

(2012) Arif Yurdagul et al.   MOLECULAR BIOLOGY OF THE CELL

Point Mutations in the ERα Gαi Binding Domain Segregate Nonnuclear from Nuclear Receptor Function

(2012) Qian Wu et al.   MOLECULAR ENDOCRINOLOGY

Pathobiology of Stent Thrombosis after Drug-Eluting Stent Implantation

(2011) Marc Vorpahl et al.   CURRENT PHARMACEUTICAL DESIGN

Effects of Disturbed Flow on Vascular Endothelium: Pathophysiological Basis and Clinical Perspectives

(2011) Jeng-Jiann Chiu et al.   PHYSIOLOGICAL REVIEWS

Essential role of ER-α-dependent NO production in resveratrol-mediated inhibition of restenosis

(2010) Alok R. Khandelwal et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Gender-Specific Reduction in Contraction is Associated with Increased Estrogen Receptor Expression in Single Vascular Smooth Muscle Cells of Female Rat

(2010) Yukui Ma et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

The Stent-Eluting Drugs Sirolimus and Paclitaxel Suppress Healing of the Endothelium by Induction of Autophagy

(2009) Shin-ichiro Hayashi et al.   AMERICAN JOURNAL OF PATHOLOGY

Estradiol accelerates endothelial healing through the retrograde commitment of uninjured endothelium

(2008) Cédric Filipe et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

Intimal Hyperplasia in Murine Models

(2008) David Hui   CURRENT DRUG TARGETS

Late occlusive in-stent restenosis of a bare-metal stent presenting with ST-elevation anterior MI: Is restenosis better than a late stent thrombosis?

(2008) Daniela Trabattoni et al.   INTERNATIONAL JOURNAL OF CARDIOLOGY

Regulation of vascular smooth muscle cell expression and function of matrix metalloproteinases is mediated by estrogen and progesterone exposure

(2008) Oscar H. Grandas et al.   JOURNAL OF VASCULAR SURGERY