The Transcription Factor ATF4 Promotes Expression of Cell Stress Genes and Cardiomyocyte Death in a Cellular Model of Atrial Fibrillation

Interplay between P-Glycoprotein Expression and Resistance to Endoplasmic Reticulum Stressors

(2018) Milan Hano et al.   MOLECULES

IRF3 and type I interferons fuel a fatal response to myocardial infarction

(2017) Kevin R King et al.   NATURE MEDICINE

The integrated stress response

(2016) Karolina Pakos‐Zebrucka et al.   EMBO REPORTS

Electrophysiological and molecular mechanisms of paroxysmal atrial fibrillation

(2016) Stanley Nattel et al.   Nature Reviews Cardiology

Defining the major health modifiers causing atrial fibrillation: a roadmap to underpin personalized prevention and treatment

(2015) Larissa Fabritz et al.   Nature Reviews Cardiology

The ER Stress-Mediated Mitochondrial Apoptotic Pathway and MAPKs Modulate Tachypacing-Induced Apoptosis in HL-1 Atrial Myocytes

(2015) Jiaojiao Shi et al.   PLoS One

A mathematical model of the unfolded protein stress response reveals the decision mechanism for recovery, adaptation and apoptosis

(2013) Kamil Erguler et al.   BMC Systems Biology

Transforming growth factor-β and oxidative stress mediate tachycardia-induced cellular remodelling in cultured atrial-derived myocytes

(2011) Yung-Hsin Yeh et al.   CARDIOVASCULAR RESEARCH

Biology of Endoplasmic Reticulum Stress in the Heart

(2010) Jody Groenendyk et al.   CIRCULATION RESEARCH

Atrial Remodeling and Atrial Fibrillation

(2008) Stanley Nattel et al.   Circulation-Arrhythmia and Electrophysiology

ATF4 is an oxidative stress–inducible, prodeath transcription factor in neurons in vitro and in vivo

(2008) Philipp S. Lange et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Activating transcription factor 4

(2007) Kurosh Ameri et al.   INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY

The role of the unfolded protein response in the heart

(2007) Christopher C. Glembotski   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY