CC16 levels correlate with cigarette smoke exposure in bronchial epithelial cells and with lung function decline in smokers
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Title
CC16 levels correlate with cigarette smoke exposure in bronchial epithelial cells and with lung function decline in smokers
Authors
Keywords
Biomarkers, Lung function, Smoking, Spirometry, Forced expiratory volume
Journal
BMC Pulmonary Medicine
Volume 18, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-04-04
DOI
10.1186/s12890-018-0607-7
References
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Related references
Note: Only part of the references are listed.- Nicotinic acetylcholine receptor expression in human airway correlates with lung function
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- Club Cell Protein 16 (CC16) Augmentation: A Potential Disease-modifying Approach for Chronic Obstructive Pulmonary Disease (COPD)
- (2016) Maria E. Laucho-Contreras et al. EXPERT OPINION ON THERAPEUTIC TARGETS
- Club cells and CC16: another “smoking gun”? (With potential bullets against COPD)
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- Low plasma CC16 levels in smokers are associated with a higher risk for chronic bronchitis: TABLE 1
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- Protective role for club cell secretory protein-16 (CC16) in the development of COPD
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- Relation between circulating CC16 concentrations, lung function, and development of chronic obstructive pulmonary disease across the lifespan: a prospective study
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- Repression of CC16 by Cigarette Smoke (CS) Exposure
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- Club Cell Protein 16 and Disease Progression in Chronic Obstructive Pulmonary Disease
- (2013) Hye Yun Park et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Spirometric Predictors of Lung Function Decline and Mortality in Early Chronic Obstructive Pulmonary Disease
- (2012) M. Bradley Drummond et al. AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE
- Inflammatory Biomarkers Improve Clinical Prediction of Mortality in Chronic Obstructive Pulmonary Disease
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- CC-16 as a Biomarker in Chronic Obstructive Pulmonary Disease
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- The Role of MAPK and Nrf2 Pathways in Ketanserin-Elicited Attenuation of Cigarette Smoke–Induced IL-8 Production in Human Bronchial Epithelial Cells
- (2011) Way Kwok Wai Lau et al. TOXICOLOGICAL SCIENCES
- Fructose Induces the Inflammatory Molecule ICAM-1 in Endothelial Cells
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- Variability of the Prevalence of Undiagnosed Airflow Obstruction in Smokers Using Different Diagnostic Criteria
- (2007) Arthur Chun-Wing Lau et al. CHEST
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