Intestinal fibrosis is associated with lack of response to Infliximab therapy in Crohn's disease

Tissue stiffness regulates serine/arginine-rich protein-mediated splicing of the extra domain B-fibronectin isoform in tumors

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Crohn's disease complicated by strictures: a systematic review

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Mechanisms of fibrosis: therapeutic translation for fibrotic disease

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Epidemiology and Natural History of Inflammatory Bowel Diseases

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Antibodies Against Tumor Necrosis Factor (TNF) Induce T-Cell Apoptosis in Patients With Inflammatory Bowel Diseases via TNF Receptor 2 and Intestinal CD14+ Macrophages

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Anti–Tumor Necrosis Factor-α Antibodies Induce Regulatory Macrophages in an Fc Region-Dependent Manner

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Predictive value of epithelial gene expression profiles for response to infliximab in Crohnʼs disease‡

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Cellular and molecular mechanisms of fibrosis

(2007) TA Wynn   JOURNAL OF PATHOLOGY