SubID, a non-median dichotomization tool for heterogeneous populations, reveals the pan-cancer significance of INPP4B and its regulation by EVI1 in AML

Biological and clinical significance of loss of heterozygosity at the INPP4B gene locus in Japanese breast cancer

(2016) Eriko Tokunaga et al.   BREAST

Genomic analyses identify molecular subtypes of pancreatic cancer

(2016) Peter Bailey et al.   NATURE

Inositol polyphosphate 4-phosphatase II (INPP4B) is associated with chemoresistance and poor outcome in AML

(2015) Sewa Rijal et al.   BLOOD

INPP4B overexpression is associated with poor clinical outcome and therapy resistance in acute myeloid leukemia

(2015) I Dzneladze et al.   LEUKEMIA

Phosphoinositide signaling in cancer: INPP4B Akt(s) out

(2015) John F. Woolley et al.   TRENDS IN MOLECULAR MEDICINE

INPP4B Is a PtdIns(3,4,5)P3 Phosphatase That Can Act as a Tumor Suppressor

(2015) S. Kofuji et al.   Cancer Discovery

INPP4B suppresses prostate cancer cell invasion

(2014) Myles C Hodgson et al.   Cell Communication and Signaling

SGK3 Mediates INPP4B-Dependent PI3K Signaling in Breast Cancer

(2014) Jessica A. Gasser et al.   MOLECULAR CELL

A Tumor Suppressor Function for the Lipid Phosphatase INPP4B in Melanocytic Neoplasms

(2013) Rolando Perez-Lorenzo et al.   JOURNAL OF INVESTIGATIVE DERMATOLOGY

A survey of immunohistochemical biomarkers for basal-like breast cancer against a gene expression profile gold standard

(2013) Jennifer R Won et al.   MODERN PATHOLOGY

Stem cell gene expression programs influence clinical outcome in human leukemia

(2011) Kolja Eppert et al.   NATURE MEDICINE

Classification and prognosis of invasive breast cancer: from morphology to molecular taxonomy

(2010) Stuart J Schnitt   MODERN PATHOLOGY

Inositol polyphosphate 4-phosphatase II regulates PI3K/Akt signaling and is lost in human basal-like breast cancers

(2010) C. G. Fedele et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Evidence that Inositol Polyphosphate 4-Phosphatase Type II Is a Tumor Suppressor that Inhibits PI3K Signaling

(2009) Christina Gewinner et al.   CANCER CELL

PI(3,4,5)P3 and PI(3,4)P2 levels correlate with PKB/akt phosphorylation at Thr308 and Ser473, respectively; PI(3,4)P2 levels determine PKB activity

(2007) Kewei Ma et al.   CELLULAR SIGNALLING