Inflammatory Cytokine, IL-1β, Regulates Glial Glutamate Transporter via microRNA-181a in vitro

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Profile of 6 microRNA in blood plasma distinguish early stage Alzheimer’s disease patients from non-demented subjects

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Identification of miRNA Changes in Alzheimer's Disease Brain and CSF Yields Putative Biomarkers and Insights into Disease Pathways

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Purification and Characterization of Progenitor and Mature Human Astrocytes Reveals Transcriptional and Functional Differences with Mouse

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Restored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer’s disease

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Ceftriaxone ameliorates tau pathology and cognitive decline via restoration of glial glutamate transporter in a mouse model of Alzheimer's disease

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Synapse-specific IL-1 receptor subunit reconfiguration augments vulnerability to IL-1β in the aged hippocampus

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The emerging role of microRNAs in Alzheimer's disease

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Differential Regulation of Glutamate Transporter Subtypes by Pro-Inflammatory Cytokine TNF-α in Cortical Astrocytes from a Rat Model of Amyotrophic Lateral Sclerosis

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Neuronal Exosomal miRNA-dependent Translational Regulation of Astroglial Glutamate Transporter GLT1

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Inhibition of microRNA-181 Reduces Forebrain Ischemia-Induced Neuronal Loss

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Amyloid- 1-42 Slows Clearance of Synaptically Released Glutamate by Mislocalizing Astrocytic GLT-1

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Relationship between increase in astrocytic GLT-1 glutamate transport and late-LTP

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Modulating the Strength and Threshold of NOTCH Oncogenic Signals by mir-181a-1/b-1

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Astrocytes support hippocampal-dependent memory and long-term potentiation via interleukin-1 signaling

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Dendritic Function of Tau Mediates Amyloid-β Toxicity in Alzheimer's Disease Mouse Models

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Activation of Extrasynaptic, But Not Synaptic, NMDA Receptors Modifies Amyloid Precursor Protein Expression Pattern and Increases Amyloid-  Production

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microRNAs at the synapse

(2009) Gerhard Schratt   NATURE REVIEWS NEUROSCIENCE

Presynaptic Regulation of Astroglial Excitatory Neurotransmitter Transporter GLT1

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Cell-specific and concentration-dependent actions of interleukin-1 in acute brain inflammation

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