Ginsenoside Rh2 inhibits prostate cancer cell growth through suppression of microRNA-4295 that activates CDKN1A

Ginsenoside-Rh2 Inhibits Proliferation and Induces Apoptosis of Human Gastric Cancer SGC-7901 Side Population Cells

(2016) Jun Qian et al.   Asian Pacific Journal of Cancer Prevention

RUNX3 is down-regulated in glioma by Myc-regulated miR-4295

(2016) Xinxing Li et al.   JOURNAL OF CELLULAR AND MOLECULAR MEDICINE

MicroRNA Target Recognition: Insights from Transcriptome-Wide Non-Canonical Interactions

(2016)   MOLECULES AND CELLS

Anti-Inflammatory Effects of Ginsenoside-Rh2 Inhibits LPS-Induced Activation of Microglia and Overproduction of Inflammatory Mediators Via Modulation of TGF-β1/Smad Pathway

(2016) R Vinoth Kumar et al.   NEUROCHEMICAL RESEARCH

XIAP RING domain mediates miR-4295 expression and subsequently inhibiting p63α protein translation and promoting transformation of bladder epithelial cells

(2016) Honglei Jin et al.   Oncotarget

Mitotic p21Cip1/CDKN1A is regulated by cyclin-dependent kinase 1 phosphorylation

(2016) Nina-Naomi Kreis et al.   Oncotarget

Ginsenoside Rh2 inhibiting HCT116 colon cancer cell proliferation through blocking PDZ-binding kinase/T-LAK cell-originated protein kinase

(2016) Jianjun Yang et al.   Journal of Ginseng Research

miR-4295 promotes cell proliferation and invasion in anaplastic thyroid carcinoma via CDKN1A

(2015) Mingchen Shao et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Prostate cancer stroma: an important factor in cancer growth and progression

(2015) Božo Krušlin et al.   Bosnian Journal of Basic Medical Sciences

Ginsenoside Rh2 Suppresses Neovascularization in Xenograft Psoriasis Model

(2015) Jing Zhou et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

Ginsenoside Rh2 Inhibits Cancer Stem-Like Cells in Skin Squamous Cell Carcinoma

(2015) Shunli Liu et al.   CELLULAR PHYSIOLOGY AND BIOCHEMISTRY

The hypoxia-induced microRNA-130a controls pulmonary smooth muscle cell proliferation by directly targeting CDKN1A

(2015) Matthias Brock et al.   INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY

Adiponectin inhibits VEGF-A in prostate cancer cells

(2015) Qiruo Gao et al.   TUMOR BIOLOGY

Predicting effective microRNA target sites in mammalian mRNAs

(2015) Vikram Agarwal et al.   eLife

Biology of the cell cycle inhibitor p21CDKN1A: molecular mechanisms and relevance in chemical toxicology

(2014) Ilaria Dutto et al.   ARCHIVES OF TOXICOLOGY

Inhibition of prostatic cancer growth by ginsenoside Rh2

(2014) Qingchuan Zhang et al.   TUMOR BIOLOGY

AMP-activated protein kinase determines apoptotic sensitivity of cancer cells to ginsenoside-Rh2

(2014) Min-Jung Kim et al.   Journal of Ginseng Research

Sulfated Derivative of 20(S)-Ginsenoside Rh2 Inhibits Inflammatory Cytokines Through MAPKs and NF-kappa B Pathways in LPS-Induced RAW264.7 Macrophages

(2012) Wen-Yan Bi et al.   INFLAMMATION

Ginsenoside Rh2 mediates changes in the microRNA expression profile of human non-small cell lung cancer A549 cells

(2012) IN-SOOK AN et al.   ONCOLOGY REPORTS

Recent Advances in Understanding Hormonal Therapy Resistant Prostate Cancer

(2010) K.V. Donkena et al.   CURRENT CANCER DRUG TARGETS

Androgen-responsive and -unresponsive prostate cancer cell lines respond differently to stimuli inducing neuroendocrine differentiation

(2010) S. Marchiani et al.   INTERNATIONAL JOURNAL OF ANDROLOGY

MicroRNA-124: Micromanager of Neurogenesis

(2009) Thales Papagiannakopoulos et al.   Cell Stem Cell

Ginsenoside Rh2-mediated G1 Phase Cell Cycle Arrest in Human Breast Cancer Cells Is Caused by p15 Ink4B and p27 Kip1 -dependent Inhibition of Cyclin-dependent Kinases

(2009) Sunga Choi et al.   PHARMACEUTICAL RESEARCH

MicroRNA Regulation of Stem Cell Fate

(2008) Qintong Li et al.   Cell Stem Cell

MicroRNA Regulation of Cell Lineages in Mouse and Human Embryonic Stem Cells

(2008) Kathryn N. Ivey et al.   Cell Stem Cell

A search for conserved sequences in coding regions reveals that the let-7 microRNA targets Dicer within its coding sequence

(2008) J. J. Forman et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA