Selenium ameliorates Staphylococcus aureus-induced inflammation in bovine mammary epithelial cells by inhibiting activation of TLR2, NF-κB and MAPK signaling pathways
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Title
Selenium ameliorates Staphylococcus aureus-induced inflammation in bovine mammary epithelial cells by inhibiting activation of TLR2, NF-κB and MAPK signaling pathways
Authors
Keywords
Se, TLR2, NF-κB, MAPK, Mammary
Journal
BMC Veterinary Research
Volume 14, Issue 1, Pages -
Publisher
Springer Nature
Online
2018-06-20
DOI
10.1186/s12917-018-1508-y
References
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Note: Only part of the references are listed.- Selenium Inhibits LPS-Induced Pro-inflammatory Gene Expression by Modulating MAPK and NF-κB Signaling Pathways in Mouse Mammary Epithelial Cells in Primary Culture
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- Comparative Kinetics ofEscherichia coli- andStaphylococcus aureus-Specific Activation of Key Immune Pathways in Mammary Epithelial Cells Demonstrates ThatS. aureusElicits a Delayed Response Dominated by Interleukin-6 (IL-6) but Not by IL-1A or Tumor Necrosis Factor Alpha
- (2010) Juliane Günther et al. INFECTION AND IMMUNITY
- Involvement of ERK1/2 signalling and growth-related molecules’ expression in response to heat stress-induced damage in rat jejunum and IEC-6 cells
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- Mastitis associated transcriptomic disruptions in cattle
- (2010) Manuela Rinaldi et al. VETERINARY IMMUNOLOGY AND IMMUNOPATHOLOGY
- Stimulated expression of TNF-α and IL-8, but not of lingual antimicrobial peptide reflects the concentration of pathogens contacting bovine mammary epithelial cells
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- Bovine TLR2 and TLR4 properly transduce signals from Staphylococcus aureus and E. coli, but S. aureus fails to both activate NF-κB in mammary epithelial cells and to quickly induce TNFα and interleukin-8 (CXCL8) expression in the udder
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