Glutathione Fine-Tunes the Innate Immune Response toward Antiviral Pathways in a Macrophage Cell Line Independently of Its Antioxidant Properties
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Title
Glutathione Fine-Tunes the Innate Immune Response toward Antiviral Pathways in a Macrophage Cell Line Independently of Its Antioxidant Properties
Authors
Keywords
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Journal
Frontiers in Immunology
Volume 8, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2017-09-29
DOI
10.3389/fimmu.2017.01239
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- (2014) David Olagnier et al. PLoS Pathogens
- Reactive Oxygen Species in Inflammation and Tissue Injury
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- (2008) M. Kobayashi et al. MOLECULAR AND CELLULAR BIOLOGY
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