Reversible Axonal Dystrophy by Calcium Modulation in Frataxin-Deficient Sensory Neurons of YG8R Mice
Published 2017 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
Reversible Axonal Dystrophy by Calcium Modulation in Frataxin-Deficient Sensory Neurons of YG8R Mice
Authors
Keywords
-
Journal
Frontiers in Molecular Neuroscience
Volume 10, Issue -, Pages -
Publisher
Frontiers Media SA
Online
2017-08-30
DOI
10.3389/fnmol.2017.00264
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Two different pathogenic mechanisms, dying-back axonal neuropathy and pancreatic senescence, are present in the YG8R mouse model of Friedreich’s ataxia
- (2016) Belén Mollá et al. Disease Models & Mechanisms
- Mitochondrial dysfunction induced by frataxin deficiency is associated with cellular senescence and abnormal calcium metabolism
- (2014) Arantxa Bolinches-Amorós et al. Frontiers in Cellular Neuroscience
- Axonal degeneration in the peripheral nervous system: Implications for the pathogenesis of amyotrophic lateral sclerosis
- (2013) Lindsey R. Fischer-Hayes et al. EXPERIMENTAL NEUROLOGY
- Apoptotic cell death and altered calcium homeostasis caused by frataxin depletion in dorsal root ganglia neurons can be prevented by BH4 domain of Bcl-xL protein
- (2013) S. Mincheva-Tasheva et al. HUMAN MOLECULAR GENETICS
- Nikolaus Friedreich and degenerative atrophy of the dorsal columns of the spinal cord
- (2013) Arnulf H. Koeppen JOURNAL OF NEUROCHEMISTRY
- Friedreich Ataxia: Neuropathology Revised
- (2013) Arnulf H. Koeppen et al. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
- Role of calpains in the injury-induced dysfunction and degeneration of the mammalian axon
- (2013) Marek Ma NEUROBIOLOGY OF DISEASE
- Cooperative role of RanBP9 and P73 in mitochondria-mediated apoptosis
- (2013) T Liu et al. Cell Death & Disease
- Focal Increases of Axoplasmic Ca2+, Aggregation of Sodium-Calcium Exchanger, N-type Ca2+ Channel, and Actin Define the Sites of Spheroids in Axons Undergoing Oxidative Stress
- (2012) A. G. Barsukova et al. JOURNAL OF NEUROSCIENCE
- A reversible form of axon damage in experimental autoimmune encephalomyelitis and multiple sclerosis
- (2011) Ivana Nikić et al. NATURE MEDICINE
- Differential Expression of PGC-1α and Metabolic Sensors Suggest Age-Dependent Induction of Mitochondrial Biogenesis in Friedreich Ataxia Fibroblasts
- (2011) José Luis García-Giménez et al. PLoS One
- Pathology and pathogenesis of sensory neuropathy in Friedreich’s ataxia
- (2010) Jennifer A. Morral et al. ACTA NEUROPATHOLOGICA
- The Neuropathology of Late-Onset Friedreich’s Ataxia
- (2010) Arnulf H. Koeppen et al. CEREBELLUM
- Frataxin and Mitochondrial FeS Cluster Biogenesis
- (2010) Timothy L. Stemmler et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Mechanisms of Axonal Spheroid Formation in Central Nervous System Wallerian Degeneration
- (2010) Bogdan Beirowski et al. JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY
- Defects in Mitochondrial Axonal Transport and Membrane Potential without Increased Reactive Oxygen Species Production in a Drosophila Model of Friedreich Ataxia
- (2010) Y. Shidara et al. JOURNAL OF NEUROSCIENCE
- Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission
- (2009) Ruben K. Dagda et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Severely dystrophic axons at amyloid plaques remain continuous and connected to viable cell bodies
- (2008) Robert Adalbert et al. BRAIN
Find the ideal target journal for your manuscript
Explore over 38,000 international journals covering a vast array of academic fields.
SearchBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started