BTG2 is a tumor suppressor gene upregulated by p53 and PTEN in human bladder carcinoma cells
Published 2017 View Full Article
- Home
- Publications
- Publication Search
- Publication Details
Title
BTG2 is a tumor suppressor gene upregulated by p53 and PTEN in human bladder carcinoma cells
Authors
Keywords
-
Journal
Cancer Medicine
Volume 7, Issue 1, Pages 184-195
Publisher
Wiley
Online
2017-12-14
DOI
10.1002/cam4.1263
References
Ask authors/readers for more resources
Related references
Note: Only part of the references are listed.- Gamma Irradiation Upregulates B-cell Translocation Gene 2 to Attenuate Cell Proliferation of Lung Cancer Cells Through the JNK and NF-κB Pathways
- (2017) Peihe Wang et al. ONCOLOGY RESEARCH
- Prostate-derived ets factor represses tumorigenesis and modulates epithelial-to-mesenchymal transition in bladder carcinoma cells
- (2016) Ke-Hung Tsui et al. CANCER LETTERS
- Inhibitory effects of B-cell translocation gene 2 on skin cancer cells via the Wnt/β-catenin signaling pathway
- (2016) Shou-Song Gao et al. Molecular Medicine Reports
- Upregulation of B-cell translocation gene 2 by epigallocatechin-3-gallate via p38 and ERK signaling blocks cell proliferation in human oral squamous cell carcinoma cells
- (2015) Jehn-Chuan Lee et al. CANCER LETTERS
- Inhibition of breast cancer invasion by TIS21/BTG2/Pc3-Akt1-Sp1-Nox4 pathway targeting actin nucleators, mDia genes
- (2015) J-A Choi et al. ONCOGENE
- Growth differentiation factor-15: a p53- and demethylation-upregulating gene represses cell proliferation, invasion, and tumorigenesis in bladder carcinoma cells
- (2015) Ke-Hung Tsui et al. Scientific Reports
- BTG2: A rising star of tumor suppressors (Review)
- (2014) BIJING MAO et al. INTERNATIONAL JOURNAL OF ONCOLOGY
- Topoisomerase Inhibitors Modulate Gene Expression of B-Cell Translocation Gene 2 and Prostate Specific Antigen in Prostate Carcinoma Cells
- (2014) Kun-Chun Chiang et al. PLoS One
- Cisplatin modulates B-cell translocation gene 2 to attenuate cell proliferation of prostate carcinoma cells in both p53-dependent and p53-independent pathways
- (2014) Kun-Chun Chiang et al. Scientific Reports
- Endogenous BTG2 expression stimulates migration of bladder cancer cells and correlates with poor clinical prognosis for bladder cancer patients
- (2013) N Wagener et al. BRITISH JOURNAL OF CANCER
- PTEN modulators: a patent review
- (2013) Chandra S Boosani et al. EXPERT OPINION ON THERAPEUTIC PATENTS
- Mechanisms by Which Interleukin-6 Attenuates Cell Invasion and Tumorigenesis in Human Bladder Carcinoma Cells
- (2013) Ke-Hung Tsui et al. Biomed Research International
- BTG2 suppresses cancer cell migration through inhibition of Src-FAK signaling by downregulation of reactive oxygen species generation in mitochondria
- (2012) Seo-Kyung Lim et al. CLINICAL & EXPERIMENTAL METASTASIS
- Current perspectives in bladder cancer management
- (2012) T. R. L. Griffiths et al. INTERNATIONAL JOURNAL OF CLINICAL PRACTICE
- Lack of progress in bladder cancer—what are the obstacles?
- (2012) Mark S. Soloway Nature Reviews Urology
- Raloxifene Inhibits Growth of RT4 Urothelial Carcinoma Cells via Estrogen Receptor-Dependent Induction of Apoptosis and Inhibition of Proliferation
- (2012) Kristi L. Hoffman et al. Hormones & Cancer
- Upregulation of prostate-derived Ets factor by luteolin causes inhibition of cell proliferation and cell invasion in prostate carcinoma cells
- (2011) Ke-Hung Tsui et al. INTERNATIONAL JOURNAL OF CANCER
- The mammalian anti-proliferative BTG/Tob protein family
- (2009) G. Sebastiaan Winkler JOURNAL OF CELLULAR PHYSIOLOGY
- BTG/TOB factors impact deadenylases
- (2009) Fabienne Mauxion et al. TRENDS IN BIOCHEMICAL SCIENCES
- Triiodothyronine modulates cell proliferation of human prostatic carcinoma cells by downregulation of the B-Cell translocation gene 2
- (2008) Ke-Hung Tsui et al. PROSTATE
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started