Peptide-nucleotide antibiotic Microcin C is a potent inducer of stringent response and persistence in both sensitive and producing cells

Molecular mechanisms and clinical implications of bacterial persistence

(2016) Joran Elie Michiels et al.   DRUG RESISTANCE UPDATES

Mechanisms of bacterial persistence during stress and antibiotic exposure

(2016) Alexander Harms et al.   SCIENCE

What Is the Link between Stringent Response, Endoribonuclease Encoding Type II Toxin–Antitoxin Systems and Persistence?

(2016) Bhaskar C. M. Ramisetty et al.   Frontiers in Microbiology

New insights into the regulatory mechanisms of ppGpp and DksA on Escherichia coli RNA polymerase–promoter complex

(2015) Nicola Doniselli et al.   NUCLEIC ACIDS RESEARCH

Stochastic induction of persister cells by HipA through (p)ppGpp-mediated activation of mRNA endonucleases

(2015) Elsa Germain et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Ribosome-controlled transcription termination is essential for the production of antibiotic microcin C

(2014) Inna Zukher et al.   NUCLEIC ACIDS RESEARCH

RETRACTED: (p)ppGpp Controls Bacterial Persistence by Stochastic Induction of Toxin-Antitoxin Activity

(2013) Etienne Maisonneuve et al.   CELL

Molecular Mechanism of Bacterial Persistence by HipA

(2013) Elsa Germain et al.   MOLECULAR CELL

HipA-mediated antibiotic persistence via phosphorylation of the glutamyl-tRNA-synthetase

(2013) Ilana Kaspy et al.   Nature Communications

Persister Cells

(2010) Kim Lewis   Annual Review of Microbiology

MccE Provides Resistance to Protein Synthesis Inhibitor Microcin C by Acetylating the Processed Form of the Antibiotic

(2010) Maria Novikova et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

The Mechanism of Microcin C Resistance Provided by the MccF Peptidase

(2010) Anton Tikhonov et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Escherichia coli Peptidase A, B, or N Can Process Translation Inhibitor Microcin C

(2008) T. Kazakov et al.   JOURNAL OF BACTERIOLOGY