Differential cellular responses by oncogenic levels of c-Myc expression in long-term confluent retinal pigment epithelial cells

Silencing of AP-4 inhibits proliferation, induces cell cycle arrest and promotes apoptosis in human lung cancer cells

(2016) XUANYU HU et al.   Oncology Letters

Ectopic AP4 expression induces cellular senescence via activation of p53 in long-term confluent retinal pigment epithelial cells

(2015) Yiping Wang et al.   EXPERIMENTAL CELL RESEARCH

AP4 activates cell migration and EMT mediated by p53 in MDA-MB-231 breast carcinoma cells

(2015) Shaopeng Chen et al.   MOLECULAR AND CELLULAR BIOCHEMISTRY

AP4 is required for mitogen- and c-MYC-induced cell cycle progression

(2015) Rene Jackstadt et al.   Oncotarget

Cell culture of retinal pigment epithelium: Special Issue

(2014) Bruce A. Pfeffer et al.   EXPERIMENTAL EYE RESEARCH

Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program

(2014) O. V. Leontieva et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

p53-Induced miR-15a/16-1 and AP4 Form a Double-Negative Feedback Loop to Regulate Epithelial–Mesenchymal Transition and Metastasis in Colorectal Cancer

(2013) Lei Shi et al.   CANCER RESEARCH

AP4 is a mediator of epithelial–mesenchymal transition and metastasis in colorectal cancer

(2013) Rene Jackstadt et al.   JOURNAL OF EXPERIMENTAL MEDICINE

Differential Effects on ARF Stability by Normal versus Oncogenic Levels of c-Myc Expression

(2013) Delin Chen et al.   MOLECULAR CELL

MYC, Metabolism, Cell Growth, and Tumorigenesis

(2013) C. V. Dang   Cold Spring Harbor Perspectives in Medicine

Down-Regulation of AP-4 Inhibits Proliferation, Induces Cell Cycle Arrest and Promotes Apoptosis in Human Gastric Cancer Cells

(2012) Xinghua Liu et al.   PLoS One

The overexpression of AP-4 as a prognostic indicator for gastric carcinoma

(2011) Liu Xinghua et al.   MEDICAL ONCOLOGY

Tumor Stroma-Derived TGF-β Limits Myc-Driven Lymphomagenesis via Suv39h1-Dependent Senescence

(2010) Maurice Reimann et al.   CANCER CELL

Tipping the Balance: Cdk2 Enables Myc to Suppress Senescence

(2010) P. Hydbring et al.   CANCER RESEARCH

Myc, Cdk2 and cellular senescence: Old players, new game

(2010) Stefano Campaner et al.   CELL CYCLE

The interaction between Myc and Miz1 is required to antagonize TGF -dependent autocrine signaling during lymphoma formation and maintenance

(2010) J. van Riggelen et al.   GENES & DEVELOPMENT

Cdk2 suppresses cellular senescence induced by the c-myc oncogene

(2009) Stefano Campaner et al.   NATURE CELL BIOLOGY

c-Myc Accelerates S-Phase and Requires WRN to Avoid Replication Stress

(2009) Kristin Robinson et al.   PLoS One

Phosphorylation by Cdk2 is required for Myc to repress Ras-induced senescence in cotransformation

(2009) P. Hydbring et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Modelling Myc inhibition as a cancer therapy

(2008) Laura Soucek et al.   NATURE

Apoptotic signaling by c-MYC

(2008) B Hoffman et al.   ONCOGENE

AP4 encodes a c-MYC-inducible repressor of p21

(2008) P. Jung et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA