TRPV4 mediates the Ca2+influx required for the interaction between flightless-1 and non-muscle myosin, and collagen remodeling
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Title
TRPV4 mediates the Ca2+influx required for the interaction between flightless-1 and non-muscle myosin, and collagen remodeling
Authors
Keywords
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Journal
JOURNAL OF CELL SCIENCE
Volume 130, Issue 13, Pages 2196-2208
Publisher
The Company of Biologists
Online
2017-05-20
DOI
10.1242/jcs.201665
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Note: Only part of the references are listed.- Enhanced store-operated Ca2+influx and ORAI1 expression in ventricular fibroblasts from human failing heart
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- TRPV4 Mechanosensitive Ion Channel Regulates Lipopolysaccharide-Stimulated Macrophage Phagocytosis
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- Flightless I interacts with NMMIIA to promote cell extension formation, which enables collagen remodeling
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- Ca2+Signaling in Cytoskeletal Reorganization, Cell Migration, and Cancer Metastasis
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- Inelastic behaviour of collagen networks in cell-matrix interactions and mechanosensation
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- TRPM7-Mediated Ca 2+ Signals Confer Fibrogenesis in Human Atrial Fibrillation
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- Ultra-rapid activation of TRPV4 ion channels by mechanical forces applied to cell surface β1 integrins
- (2010) Benjamin D. Matthews et al. Integrative Biology
- Identification and characterization of novel TRPV4 modulators
- (2009) Fabien Vincent et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- TRPV4 Channels Mediate Cyclic Strain–Induced Endothelial Cell Reorientation Through Integrin-to-Integrin Signaling
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- Helix Straightening as an Activation Mechanism in the Gelsolin Superfamily of Actin Regulatory Proteins
- (2009) Hui Wang et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Calcium flickers steer cell migration
- (2009) Chaoliang Wei et al. NATURE
- Actin and α-actinin orchestrate the assembly and maturation of nascent adhesions in a myosin II motor-independent manner
- (2008) Colin K. Choi et al. NATURE CELL BIOLOGY
- Traction Dynamics of Filopodia on Compliant Substrates
- (2008) C. E. Chan et al. SCIENCE
- Flightless I: An actin-remodelling protein and an important negative regulator of wound repair
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