The TRPC1 Ca2+-permeable channel inhibits exercise-induced protection against high-fat diet-induced obesity and type II diabetes

Docosahexaenoic Acid–Derived Fatty Acid Esters of Hydroxy Fatty Acids (FAHFAs) With Anti-inflammatory Properties

(2016) Ondrej Kuda et al.   DIABETES

Decreased beige adipocyte number and mitochondrial respiration coincide with increased histone methyl transferase (G9a) and reduced FGF21 gene expression in Sprague–Dawley rats fed prenatal low protein and postnatal high-fat diets

(2016) Kate J. Claycombe et al.   JOURNAL OF NUTRITIONAL BIOCHEMISTRY

A Novel Role for Subcutaneous Adipose Tissue in Exercise-Induced Improvements in Glucose Homeostasis

(2015) Kristin I. Stanford et al.   DIABETES

Store-operated Ca2+Entry Mediated by Orai1 and TRPC1 Participates to Insulin Secretion in Rat β-Cells

(2015) Jessica Sabourin et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Skeletal muscle Sirt3 expression and mitochondrial respiration are regulated by a prenatal low-protein diet

(2015) Kate J. Claycombe et al.   JOURNAL OF NUTRITIONAL BIOCHEMISTRY

Maternal low-protein diet causes body weight loss in male, neonate Sprague–Dawley rats involving UCP-1-mediated thermogenesis

(2015) Kate J. Claycombe et al.   JOURNAL OF NUTRITIONAL BIOCHEMISTRY

TRPC1-mediated Ca2+ entry is essential for the regulation of hypoxia and nutrient depletion-dependent autophagy

(2015) P Sukumaran et al.   Cell Death & Disease

Greater muscle protein synthesis and mitochondrial biogenesis in males compared with females during sprint interval training

(2014) Rebecca L. Scalzo et al.   FASEB JOURNAL

Prenatal Low-Protein and Postnatal High-Fat Diets Induce Rapid Adipose Tissue Growth by Inducing Igf2 Expression in Sprague Dawley Rat Offspring

(2013) Kate J. Claycombe et al.   JOURNAL OF NUTRITION

TRPV4 Is a Regulator of Adipose Oxidative Metabolism, Inflammation, and Energy Homeostasis

(2012) Li Ye et al.   CELL

Constitutively Active TRPC Channels of Adipocytes Confer a Mechanism for Sensing Dietary Fatty Acids and Regulating Adiponectin

(2012) Piruthivi Sukumar et al.   CIRCULATION RESEARCH

Canonical transient receptor potential channels in diabetes

(2012) Sarabeth Graham et al.   EXPERIMENTAL BIOLOGY AND MEDICINE

Non-Stimulated, Agonist-Stimulated and Store-Operated Ca2+ Influx in MDA-MB-468 Breast Cancer Cells and the Effect of EGF-Induced EMT on Calcium Entry

(2012) Felicity M. Davis et al.   PLoS One

Saturated Fatty Acids Induce c-Src Clustering within Membrane Subdomains, Leading to JNK Activation

(2011) Ryan G. Holzer et al.   CELL

Serum calcium level is associated with metabolic syndrome in the general population: FIN-D2D study

(2011) J Saltevo et al.   EUROPEAN JOURNAL OF ENDOCRINOLOGY

TRPC channels as STIM1-regulated SOCs

(2010) Joseph P. Yuan et al.   Channels

Leptin Excites Proopiomelanocortin Neurons via Activation of TRPC Channels

(2010) J. Qiu et al.   JOURNAL OF NEUROSCIENCE

Eicosapentaenoic Acid Prevents and Reverses Insulin Resistance in High-Fat Diet-Induced Obese Mice via Modulation of Adipose Tissue Inflammation

(2010) Nishan S. Kalupahana et al.   JOURNAL OF NUTRITION

Role of TRPC1 channel in skeletal muscle function

(2009) Nadège Zanou et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Effects of exercise training on subcutaneous and visceral adipose tissue in normal- and high-fat diet-fed rats

(2009) Katja S. C. Gollisch et al.   AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM

TRPC1 inhibits apoptotic cell degeneration induced by dopaminergic neurotoxin MPTP/MPP+

(2009) Senthil Selvaraj et al.   CELL CALCIUM

Lipid rafts/caveolae as microdomains of calcium signaling

(2009) Biswaranjan Pani et al.   CELL CALCIUM

Adipose Tissue Dysfunction in Obesity

(2009) M. Blüher   EXPERIMENTAL AND CLINICAL ENDOCRINOLOGY & DIABETES

Canonical Transient Receptor Potential Channels Expression Is Elevated in a Porcine Model of Metabolic Syndrome

(2009) Guoqing Hu et al.   MOLECULAR ENDOCRINOLOGY

Calcium homeostasis, selective vulnerability and Parkinson's disease

(2009) C. Savio Chan et al.   TRENDS IN NEUROSCIENCES