The complement system as a biomarker of disease activity and response to treatment in multiple sclerosis
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Title
The complement system as a biomarker of disease activity and response to treatment in multiple sclerosis
Authors
Keywords
Complement activation, RGC-32, Multiple sclerosis, Glatiramer acetate, Biomarker, Factor H
Journal
IMMUNOLOGIC RESEARCH
Volume 65, Issue 6, Pages 1103-1109
Publisher
Springer Nature
Online
2017-11-08
DOI
10.1007/s12026-017-8961-8
References
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Related references
Note: Only part of the references are listed.- Myelin-specific multiple sclerosis antibodies cause complement-dependent oligodendrocyte loss and demyelination
- (2017) Yiting Liu et al. Acta Neuropathologica Communications
- Complement Receptor 2 is increased in cerebrospinal fluid of multiple sclerosis patients and regulates C3 function
- (2016) Rickard P.F. Lindblom et al. CLINICAL IMMUNOLOGY
- Complement C3 on microglial clusters in multiple sclerosis occur in chronic but not acute disease: Implication for disease pathogenesis
- (2016) Iliana Michailidou et al. GLIA
- Plasma complement biomarkers distinguish multiple sclerosis and neuromyelitis optica spectrum disorder
- (2016) Svetlana Hakobyan et al. Multiple Sclerosis Journal
- Plasma complement biomarkers distinguish multiple sclerosis and neuromyelitis optica spectrum disorder
- (2016) Svetlana Hakobyan et al. Multiple Sclerosis Journal
- RGC-32 as a potential biomarker of relapse and response to treatment with glatiramer acetate in multiple sclerosis
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- Complement Component C3 and Butyrylcholinesterase Activity Are Associated with Neurodegeneration and Clinical Disability in Multiple Sclerosis
- (2015) Shahin Aeinehband et al. PLoS One
- Complement activation in patients with neuromyelitis optica
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- Complement activation in multiple sclerosis plaques: an immunohistochemical analysis
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- Dual role of Response gene to complement-32 in multiple sclerosis
- (2012) Cosmin A. Tegla et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- Systemic complement profiling in multiple sclerosis as a biomarker of disease state
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- C5b-9-activated, Kv1.3 channels mediate oligodendrocyte cell cycle activation and dedifferentiation
- (2011) Cosmin A. Tegla et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- Membrane attack by complement: the assembly and biology of terminal complement complexes
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- Differential expression of complement proteins in cerebrospinal fluid from active multiple sclerosis patients
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- Mannose binding lectin mediated complement pathway in multiple sclerosis
- (2011) Janet Y. Kwok et al. JOURNAL OF NEUROIMMUNOLOGY
- Complement regulator factor H as a serum biomarker of multiple sclerosis disease state
- (2010) Gillian Ingram et al. BRAIN
- Elevated plasma C4a levels in multiple sclerosis correlate with disease activity
- (2010) Gillian Ingram et al. JOURNAL OF NEUROIMMUNOLOGY
- Immunoglobulins and complement in postmortem multiple sclerosis tissue
- (2009) Michael H. Barnett et al. ANNALS OF NEUROLOGY
- Response gene to complement 32 is required for C5b-9 induced cell cycle activation in endothelial cells
- (2009) Matthew Fosbrink et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- Epigenetic modifications induced by RGC-32 in colon cancer
- (2009) Sonia I. Vlaicu et al. EXPERIMENTAL AND MOLECULAR PATHOLOGY
- Homogeneity of active demyelinating lesions in established multiple sclerosis
- (2008) Esther C. W. Breij et al. ANNALS OF NEUROLOGY
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