A mouse model reveals that Mfsd2a is critical for unfolded protein response upon exposure to tunicamycin

Inactivating mutations in MFSD2A, required for omega-3 fatty acid transport in brain, cause a lethal microcephaly syndrome

(2015) Alicia Guemez-Gamboa et al.   NATURE GENETICS

Tunicamycin-induced unfolded protein response in the developing mouse brain

(2015) Haiping Wang et al.   TOXICOLOGY AND APPLIED PHARMACOLOGY

Mfsd2a is critical for the formation and function of the blood–brain barrier

(2014) Ayal Ben-Zvi et al.   NATURE

Mfsd2a is a transporter for the essential omega-3 fatty acid docosahexaenoic acid

(2014) Long N. Nguyen et al.   NATURE

The major facilitator superfamily (MFS) revisited

(2012) Vamsee S. Reddy et al.   FEBS Journal

MFSD2a, the Syncytin-2 receptor, is important for trophoblast fusion

(2012) C. Toufaily et al.   PLACENTA

Major Facilitator Superfamily Domain-Containing Protein 2a (MFSD2A) Has Roles in Body Growth, Motor Function, and Lipid Metabolism

(2012) Justin H. Berger et al.   PLoS One

A haploid genetic screen identifies the major facilitator domain containing 2A (MFSD2A) transporter as a key mediator in the response to tunicamycin

(2011) J. H. Reiling et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

C-MYC rearrangement may induce an aggressive phenotype in anaplastic lymphoma kinase positive anaplastic large cell lymphoma: Identification of a novel fusion gene ALO17/C-MYC

(2010) Hiroshi Moritake et al.   AMERICAN JOURNAL OF HEMATOLOGY

A placenta-specific receptor for the fusogenic, endogenous retrovirus-derived, human syncytin-2

(2008) C. Esnault et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA