RhoA determines lineage fate of mesenchymal stem cells by modulating CTGF–VEGF complex in extracellular matrix
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Title
RhoA determines lineage fate of mesenchymal stem cells by modulating CTGF–VEGF complex in extracellular matrix
Authors
Keywords
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Journal
Nature Communications
Volume 7, Issue -, Pages 11455
Publisher
Springer Nature
Online
2016-04-29
DOI
10.1038/ncomms11455
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- (2014) Diana Klein et al. Frontiers in Oncology
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- Growth Differentiation Factor 11 Is a Circulating Factor that Reverses Age-Related Cardiac Hypertrophy
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- (2013) Markella Ponticos VASCULAR PHARMACOLOGY
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- (2012) Xia Guo et al. AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY
- Host Responses in Tissue Repair and Fibrosis
- (2012) Jeremy S. Duffield et al. Annual Review of Pathology-Mechanisms of Disease
- Cerebral cavernous malformation is a vascular disease associated with activated RhoA signaling
- (2012) Bryan T. Richardson et al. BIOLOGICAL CHEMISTRY
- Mechanisms of post-intervention arterial remodelling
- (2012) S. A. Goel et al. CARDIOVASCULAR RESEARCH
- Endothelial Cells Overexpressing Interleukin-8 Receptors Reduce Inflammatory and Neointimal Responses to Arterial Injury
- (2012) Dongqi Xing et al. CIRCULATION
- Myofibroblast-mediated mechanisms of pathological remodelling of the heart
- (2012) Karl T. Weber et al. Nature Reviews Cardiology
- Direct reprogramming of fibroblasts into endothelial cells capable of angiogenesis and reendothelialization in tissue-engineered vessels
- (2012) A. Margariti et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Injury-Activated Transforming Growth Factor β Controls Mobilization of Mesenchymal Stem Cells for Tissue Remodeling
- (2012) Mei Wan et al. STEM CELLS
- Effects of transforming growth factor-beta1 on cell motility, collagen gel contraction, myofibroblastic differentiation, and extracellular matrix expression of human adipose-derived stem cell
- (2012) Natsuko Kakudo et al. Human Cell
- TGFβ-induced Early Activation of the Small GTPase RhoA is Smad2/3-independent and Involves Src and the Guanine Nucleotide Exchange Factor Vav2
- (2011) Elsa Papadimitriou et al. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY
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- (2010) A. Forte et al. CARDIOVASCULAR RESEARCH
- Rho Kinase Inhibition Rescues the Endothelial Cell Cerebral Cavernous Malformation Phenotype
- (2010) Asya L. Borikova et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Mesenchymal and haematopoietic stem cells form a unique bone marrow niche
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- Control of Stem Cell Fate by Physical Interactions with the Extracellular Matrix
- (2009) Farshid Guilak et al. Cell Stem Cell
- Stem cells, microenvironment mechanics, and growth factor activation
- (2009) Rebeca M Tenney et al. CURRENT OPINION IN CELL BIOLOGY
- TGF-β1–induced migration of bone mesenchymal stem cells couples bone resorption with formation
- (2009) Yi Tang et al. NATURE MEDICINE
- Growth Factors, Matrices, and Forces Combine and Control Stem Cells
- (2009) D. E. Discher et al. SCIENCE
- lumi: a pipeline for processing Illumina microarray
- (2008) P. Du et al. BIOINFORMATICS
- MAPK signalling in cardiovascular health and disease: molecular mechanisms and therapeutic targets
- (2008) Anthony J. Muslin CLINICAL SCIENCE
- A novel mechanism of TGFβ-induced actin reorganization mediated by Smad proteins and Rho GTPases
- (2008) Lina Vardouli et al. FEBS Journal
- Parathyroid hormone signaling through low-density lipoprotein-related protein 6
- (2008) M. Wan et al. GENES & DEVELOPMENT
- Connective tissue growth factor: Structure–function relationships of a mosaic, multifunctional protein
- (2008) Patricia De Winter et al. GROWTH FACTORS
- TRAF6 Mediates Smad-Independent Activation of JNK and p38 by TGF-β
- (2008) Motozo Yamashita et al. MOLECULAR CELL
- Cell biology: Stretching the imagination
- (2008) Claire Ainsworth NATURE
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- Circulating progenitor cells contribute to neointimal formation in nonirradiated chimeric mice
- (2007) Kimie Tanaka et al. FASEB JOURNAL
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