Revisiting the truncated lamin A produced by a commonly used strain of Lmna knockout mice
Published 2023 View Full Article
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Title
Revisiting the truncated lamin A produced by a commonly used strain of
Lmna
knockout mice
Authors
Keywords
-
Journal
Nucleus
Volume 14, Issue 1, Pages -
Publisher
Informa UK Limited
Online
2023-09-27
DOI
10.1080/19491034.2023.2262308
References
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Related references
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- (2021) Paul H. Kim et al. JCI Insight
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- (2019) Ashley J. Earle et al. NATURE MATERIALS
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- Disrupting the LINC complex in smooth muscle cells reduces aortic disease in a mouse model of Hutchinson-Gilford progeria syndrome
- (2018) Paul H. Kim et al. Science Translational Medicine
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- (2014) H.-J. Jung et al. MOLECULAR AND CELLULAR BIOLOGY
- LBR and Lamin A/C Sequentially Tether Peripheral Heterochromatin and Inversely Regulate Differentiation
- (2013) Irina Solovei et al. CELL
- Farnesylation of lamin B1 is important for retention of nuclear chromatin during neuronal migration
- (2013) H.-J. Jung et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A truncated lamin A in the Lmna−/−mouse line
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- Post-natal myogenic and adipogenic developmental
- (2011) Nard Kubben et al. Nucleus
- An accumulation of non-farnesylated prelamin A causes cardiomyopathy but not progeria
- (2010) B. S. J. Davies et al. HUMAN MOLECULAR GENETICS
- Lamin A/C haploinsufficiency causes dilated cardiomyopathy and apoptosis-triggered cardiac conduction system disease
- (2007) Cordula M. Wolf et al. JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
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