MA-5 ameliorates autism-like behavior in mice prenatally exposed to valproic acid

Microglia and astrocytes underlie neuroinflammation and synaptic susceptibility in autism spectrum disorder

(2023) Yue Xiong et al.   Frontiers in Neuroscience

Controlling litter effects to enhance rigor and reproducibility with rodent models of neurodevelopmental disorders

(2021) Jessica A. Jiménez et al.   Journal of Neurodevelopmental Disorders

Emerging mechanisms of valproic acid-induced neurotoxic events in autism and its implications for pharmacological treatment

(2021) Abdoh Taleb et al.   BIOMEDICINE & PHARMACOTHERAPY

Mitochondria May Mediate Prenatal Environmental Influences in Autism Spectrum Disorder

(2021) Richard E. Frye et al.   Journal of Personalized Medicine

The Role of Mitochondria in Mood Disorders: From Physiology to Pathophysiology and to Treatment

(2021) Anna Giménez-Palomo et al.   Frontiers in Psychiatry

Prenatal exposure to valproic acid and treatment with intranasal oxytocin have sex-specific effects on behavior in Long Evans rats

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Sex specific autistic endophenotypes induced by prenatal exposure to valproic acid involve anandamide signaling

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Biomarker-Guided Strategy for Treatment of Autism Spectrum Disorder (ASD)

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Prevalence of nuclear and mitochondrial DNA mutations related to adult mitochondrial disease

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Mitochonic Acid 5 Binds Mitochondria and Ameliorates Renal Tubular and Cardiac Myocyte Damage

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Mitochonic Acid 5 (MA-5), a Derivative of the Plant Hormone Indole-3-Acetic Acid, Improves Survival of Fibroblasts from Patients with Mitochondrial Diseases

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Novel intracellular functions of apolipoproteins: the ApoO protein family as constituents of the Mitofilin/MINOS complex determines cristae morphology in mitochondria

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Evidence linking oxidative stress, mitochondrial dysfunction, and inflammation in the brain of individuals with autism

(2014) Daniel A. Rossignol et al.   Frontiers in Physiology

Mitochondrial abnormalities in temporal lobe of autistic brain

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Rational Design of an Auxin Antagonist of the SCFTIR1 Auxin Receptor Complex

(2012) Ken-ichiro Hayashi et al.   ACS Chemical Biology

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Abnormal Behavior in a Chromosome- Engineered Mouse Model for Human 15q11-13 Duplication Seen in Autism

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Cellular and mitochondrial glutathione redox imbalance in lymphoblastoid cells derived from children with autism

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Altered calcium homeostasis in autism-spectrum disorders: evidence from biochemical and genetic studies of the mitochondrial aspartate/glutamate carrier AGC1

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