Autonomous rhythmic activity in glioma networks drives brain tumour growth

Glioblastoma hijacks neuronal mechanisms for brain invasion

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p120-catenin-dependent collective brain infiltration by glioma cell networks

(2020) Pavlo G. Gritsenko et al.   NATURE CELL BIOLOGY

Repurposing the KCa3.1 inhibitor senicapoc for Alzheimer's disease

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Coding and decoding of oscillatory Ca2+ signals

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An Integrative Model of Cellular States, Plasticity, and Genetics for Glioblastoma

(2019) Cyril Neftel et al.   CELL

Electrical and synaptic integration of glioma into neural circuits

(2019) Humsa S. Venkatesh et al.   NATURE

Glutamatergic synaptic input to glioma cells drives brain tumour progression

(2019) Varun Venkataramani et al.   NATURE

Harmful networks in the brain and beyond

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Inhibition of the potassium channel K Ca 3.1 by senicapoc reverses tactile allodynia in rats with peripheral nerve injury

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Tumor microtubes convey resistance to surgical lesions and chemotherapy in gliomas

(2017) Sophie Weil et al.   NEURO-ONCOLOGY

The potassium channel KCa3.1 constitutes a pharmacological target for neuroinflammation associated with ischemia/reperfusion stroke

(2016) Yi-Je Chen et al.   JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM

A three ion channel genes-based signature predicts prognosis of primary glioblastoma patients and reveals a chemotherapy sensitive subtype

(2016) Hao-Yuan Wang et al.   Oncotarget

KCa3.1 channel inhibition sensitizes malignant gliomas to temozolomide treatment

(2016) Giuseppina D'Alessandro et al.   Oncotarget

Brain tumour cells interconnect to a functional and resistant network

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Glioma

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A proinvasive role for the Ca2+-activated K+channel KCa3.1 in malignant glioma

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Frequency decoding of calcium oscillations

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SK4 Ca2+ activated K+ channel is a critical player in cardiac pacemaker derived from human embryonic stem cells

(2013) D. Weisbrod et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Cancer Genome Landscapes

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KCa3.1 channels are involved in the infiltrative behavior of glioblastoma in vivo

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The Inhibition of KCa3.1 Channels Activity Reduces Cell Motility in Glioblastoma Derived Cancer Stem Cells

(2012) Paola Ruggieri et al.   PLoS One

Improvements in haemolysis and indicators of erythrocyte survival do not correlate with acute vaso-occlusive crises in patients with sickle cell disease: a phase III randomized, placebo-controlled, double-blind study of the gardos channel blocker senicapo

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Decoding cytosolic Ca2+ oscillations

(2010) Anant B. Parekh   TRENDS IN BIOCHEMICAL SCIENCES

Calcium Oscillations

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Mechanisms underlying spontaneous patterned activity in developing neural circuits

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