MALAT1 induces tongue cancer cells' EMT and inhibits apoptosis through Wnt/β-catenin signaling pathway
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Title
MALAT1 induces tongue cancer cells' EMT and inhibits apoptosis through Wnt/β-catenin signaling pathway
Authors
Keywords
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Journal
JOURNAL OF ORAL PATHOLOGY & MEDICINE
Volume 46, Issue 2, Pages 98-105
Publisher
Wiley
Online
2016-06-29
DOI
10.1111/jop.12466
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Related references
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- (2014) Zhaoyu Lin et al. CANCER SCIENCE
- TGF- -Induced Upregulation of malat1 Promotes Bladder Cancer Metastasis by Associating with suz12
- (2014) Y. Fan et al. CLINICAL CANCER RESEARCH
- Long Non-Coding RNA in Cancer
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- MALAT1 — a paradigm for long noncoding RNA function in cancer
- (2013) Tony Gutschner et al. JOURNAL OF MOLECULAR MEDICINE-JMM
- PAF and EZH2 Induce Wnt/β-Catenin Signaling Hyperactivation
- (2013) Hae-Yun Jung et al. MOLECULAR CELL
- Upregulated MALAT-1 contributes to bladder cancer cell migration by inducing epithelial-to-mesenchymal transition
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- Inducing Cell Proliferation Inhibition, Apoptosis, and Motility Reduction by Silencing Long Noncoding Ribonucleic Acid Metastasis-associated Lung Adenocarcinoma Transcript 1 in Urothelial Carcinoma of the Bladder
- (2012) Yonghua Han et al. UROLOGY
- Foxp3 expressed by tongue squamous cell carcinoma cells correlates with clinicopathologic features and overall survival in tongue squamous cell carcinoma patients
- (2011) Yu-jie Liang et al. ORAL ONCOLOGY
- Inhibition of metastasis-associated lung adenocarcinoma transcript 1 in CaSki human cervical cancer cells suppresses cell proliferation and invasion
- (2010) F. Guo et al. ACTA BIOCHIMICA ET BIOPHYSICA SINICA
- MALAT-1 enhances cell motility of lung adenocarcinoma cells by influencing the expression of motility-related genes
- (2010) Keiko Tano et al. FEBS LETTERS
- Involvement of the Wnt-β-catenin pathway in invasion and migration of oral squamous carcinoma cells
- (2010) Iwai INTERNATIONAL JOURNAL OF ONCOLOGY
- A transcriptional sketch of a primary human breast cancer by 454 deep sequencing
- (2009) Alessandro Guffanti et al. BMC GENOMICS
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