Complement is activated in progressive multiple sclerosis cortical grey matter lesions
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Title
Complement is activated in progressive multiple sclerosis cortical grey matter lesions
Authors
Keywords
Complement, Grey matter lesion, Innate immunity, Multiple sclerosis, Neurodegeneration
Journal
Journal of Neuroinflammation
Volume 13, Issue 1, Pages -
Publisher
Springer Nature
Online
2016-06-23
DOI
10.1186/s12974-016-0611-x
References
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- (2012) G Ingram et al. Multiple Sclerosis Journal
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- Modulation of Tau Phosphorylation by the Kinase PKR: Implications in Alzheimer's Disease
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- (2009) Josa M. Frischer et al. BRAIN
- C1q Differentially Modulates Phagocytosis and Cytokine Responses during Ingestion of Apoptotic Cells by Human Monocytes, Macrophages, and Dendritic Cells
- (2009) D. A. Fraser et al. JOURNAL OF IMMUNOLOGY
- C1q enhances microglial clearance of apoptotic neurons and neuronal blebs, and modulates subsequent inflammatory cytokine production
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- (2008) Birgit S Budde et al. NATURE GENETICS
- TDP-43 regulates retinoblastoma protein phosphorylation through the repression of cyclin-dependent kinase 6 expression
- (2008) Y. M. Ayala et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
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