WNT10A promotes an invasive and self-renewing phenotype in esophageal squamous cell carcinoma

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Identification of genomic alterations in oesophageal squamous cell cancer

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Causal analysis approaches in Ingenuity Pathway Analysis

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Matricellular proteins: priming the tumour microenvironment for cancer development and metastasis

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Wnt signaling in stem and cancer stem cells

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Periostin cooperates with mutant p53 to mediate invasion through the induction of STAT1 signaling in the esophageal tumor microenvironment

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A uniform human Wnt expression library reveals a shared secretory pathway and unique signaling activities

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Optical Imaging of Periostin Enables Early Endoscopic Detection and Characterization of Esophageal Cancer in Mice

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NIH Image to ImageJ: 25 years of image analysis

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Isolation and characterization of mouse and human esophageal epithelial cells in 3D organotypic culture

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WNT10A Plays an Oncogenic Role in Renal Cell Carcinoma by Activating WNT/β-catenin Pathway

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Wnt Proteins

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Wnt Signaling in Cancer

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Deletion of p120-Catenin Results in a Tumor Microenvironment with Inflammation and Cancer that Establishes It as a Tumor Suppressor Gene

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Interactions between cancer stem cells and their niche govern metastatic colonization

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Stabilization of phenotypic plasticity through mesenchymal-specific DNA hypermethylation in cancer cells

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Proteomic profiling of secretome and adherent plasma membranes from distinct mammary epithelial cell subpopulations

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Periostin, a Cell Adhesion Molecule, Facilitates Invasion in the Tumor Microenvironment and Annotates a Novel Tumor-Invasive Signature in Esophageal Cancer

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Only four genes (EDA1, EDAR, EDARADD, and WNT10A) account for 90% of hypohidrotic/anhidrotic ectodermal dysplasia cases

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WNT10A Mutations Are a Frequent Cause of a Broad Spectrum of Ectodermal Dysplasias with Sex-Biased Manifestation Pattern in Heterozygotes

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Establishment of Intestinal Identity and Epithelial-Mesenchymal Signaling by Cdx2

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Wnt/β-Catenin Signaling: Components, Mechanisms, and Diseases

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WNT10A missense mutation associated with a complete Odonto-Onycho-Dermal Dysplasia syndrome

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SOX2 is an amplified lineage-survival oncogene in lung and esophageal squamous cell carcinomas

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The Epithelial-Mesenchymal Transition Generates Cells with Properties of Stem Cells

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