Impact ofStaphylococcus aureusUSA300 Colonization and Skin Infections on Systemic Immune Responses in Humans

Nonredundant Roles of Interleukin-17A (IL-17A) and IL-22 in Murine Host Defense against Cutaneous and Hematogenous Infection Due to Methicillin-Resistant Staphylococcus aureus

(2015) Liana C. Chan et al.   INFECTION AND IMMUNITY

Genetic Variation inTLR10, an Inhibitory Toll-Like Receptor, Influences Susceptibility to Complicated Skin and Skin Structure Infections

(2015) Mark H. T. Stappers et al.   JOURNAL OF INFECTIOUS DISEASES

The Fall of a Dogma? Unexpected High T-Cell Memory Response toStaphylococcus aureusin Humans

(2015) Julia B. Kolata et al.   JOURNAL OF INFECTIOUS DISEASES

Clindamycin versus Trimethoprim–Sulfamethoxazole for Uncomplicated Skin Infections

(2015) Loren G. Miller et al.   NEW ENGLAND JOURNAL OF MEDICINE

Polymorphisms in cytokine genes IL6, TNF, IL10, IL17A and IFNG influence susceptibility to complicated skin and skin structure infections

(2014) M. H. T. Stappers et al.   EUROPEAN JOURNAL OF CLINICAL MICROBIOLOGY & INFECTIOUS DISEASES

Protective Immunity against Recurrent Staphylococcus aureus Skin Infection Requires Antibody and Interleukin-17A

(2014) Christopher P. Montgomery et al.   INFECTION AND IMMUNITY

High Staphylococcus aureus Colonization Prevalence among Patients with Skin and Soft Tissue Infections and Controls in an Urban Emergency Department

(2014) Neha Kumar et al.   JOURNAL OF CLINICAL MICROBIOLOGY

Dialogue between skin microbiota and immunity

(2014) Y. Belkaid et al.   SCIENCE

Molecular mechanisms of mucocutaneous immunity against Candida and Staphylococcus species

(2012) László Maródi et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

Comparing Pulsed-Field Gel Electrophoresis with Multilocus Sequence Typing,spaTyping, Staphylococcal Cassette Chromosomemec(SCCmec) Typing, and PCR for Panton-Valentine Leukocidin,arcA, andopp3in Methicillin-Resistant Staphylococcus aureus Isolates at a U.S. Medical Center

(2012) Michael Z. David et al.   JOURNAL OF CLINICAL MICROBIOLOGY

Pathogen-induced human TH17 cells produce IFN-γ or IL-10 and are regulated by IL-1β

(2012) Christina E. Zielinski et al.   NATURE

IL-17 is essential for host defense against cutaneous Staphylococcus aureus infection in mice

(2010) John S. Cho et al.   JOURNAL OF CLINICAL INVESTIGATION

Deficiency of IL-22 Contributes to a Chronic Inflammatory Disease: Pathogenetic Mechanisms in Acne Inversa

(2010) K. Wolk et al.   JOURNAL OF IMMUNOLOGY

Immunological surrogate marker of rAls3p-N vaccine-induced protection againstStaphylococcus aureus: Table 1

(2009) Lin Lin et al.   FEMS IMMUNOLOGY AND MEDICAL MICROBIOLOGY

Differential Roles of Interleukin-17A and -17F in Host Defense against Mucoepithelial Bacterial Infection and Allergic Responses

(2009) Harumichi Ishigame et al.   IMMUNITY

Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome

(2009) Yoshiyuki Minegishi et al.   JOURNAL OF EXPERIMENTAL MEDICINE

NOD2 contributes to cutaneous defense against Staphylococcus aureus through  -toxin-dependent innate immune activation

(2009) P. Hruz et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

The Antifungal Vaccine Derived from the Recombinant N Terminus of Als3p Protects Mice against the Bacterium Staphylococcus aureus

(2008) B. Spellberg et al.   INFECTION AND IMMUNITY

Novel signal transducer and activator of transcription 3 (STAT3) mutations, reduced TH17 cell numbers, and variably defective STAT3 phosphorylation in hyper-IgE syndrome

(2008) Ellen D. Renner et al.   JOURNAL OF ALLERGY AND CLINICAL IMMUNOLOGY

Deficiency of Th17 cells in hyper IgE syndrome due to mutations inSTAT3

(2008) Cindy S. Ma et al.   JOURNAL OF EXPERIMENTAL MEDICINE

What Is Community‐Associated Methicillin‐ResistantStaphylococcus aureus?

(2008) Michael Z. David et al.   JOURNAL OF INFECTIOUS DISEASES

Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome

(2008) Joshua D. Milner et al.   NATURE